Hasil Pencarian - Vittorio Rosti

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  1. 1
    Therapeutic Potential of Endothelial Colony-Forming Cells in Ischemic Disease: Strategies to Improve their Regenerative Efficacy

    Therapeutic Potential of Endothelial Colony-Forming Cells in Ischemic Disease: Strategies to Improve their Regenerative Efficacy oleh Pawan Faris, Sharon Negri, Angelica Perna, Vittorio Rosti, Germano Guerra, Francesco Moccia

    Diterbitkan 2020-10-01
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  2. 2
    Vascular endothelial growth factor promoted endothelial progenitor cell mobilization into the peripheral blood of a patient with POEMS syndrome

    Vascular endothelial growth factor promoted endothelial progenitor cell mobilization into the peripheral blood of a patient with POEMS syndrome oleh Vittorio Rosti, Margherita Massa, Rita Campanelli, Mara De Amici, Giovanni Piccolo, Vittorio Perfetti

    Diterbitkan 2007-09-01
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  3. 3
    Endothelial TRPV1 as an Emerging Molecular Target to Promote Therapeutic Angiogenesis

    Endothelial TRPV1 as an Emerging Molecular Target to Promote Therapeutic Angiogenesis oleh Sharon Negri, Pawan Faris, Vittorio Rosti, Maria Rosa Antognazza, Francesco Lodola, Francesco Moccia

    Diterbitkan 2020-05-01
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  4. 4
    Elevated plasma EDA fibronectin in primary myelofibrosis is determined by high allele burden of JAK2V617F mutation and strongly predicts splenomegaly progression

    Elevated plasma EDA fibronectin in primary myelofibrosis is determined by high allele burden of JAK2V617F mutation and strongly predicts splenomegaly progression oleh Alessandro Malara, Cristian Gruppi, Margherita Massa, Maria Enrica Tira, Vittorio Rosti, Alessandra Balduini, Giovanni Barosi

    Diterbitkan 2022-09-01
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  5. 5
    Type 1 CALR mutation allele frequency correlates with CD34/CXCR4 expression in myelofibrosis-type megakaryocyte dysplasia: A mechanism of disease progression?

    Type 1 CALR mutation allele frequency correlates with CD34/CXCR4 expression in myelofibrosis-type megakaryocyte dysplasia: A mechanism of disease progression? oleh Giovanni Barosi, Rita Campanelli, Paolo Catarsi, Carlotta Abbà, Adriana Carolei, Margherita Massa, Robert Peter Gale, Vittorio Rosti

    Diterbitkan 2024-01-01
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  6. 6
    No association between the XPD Lys751Gln (rs13181) polymorphism and disease phenotype or leukemic transformation in primary myelofibrosis

    No association between the XPD Lys751Gln (rs13181) polymorphism and disease phenotype or leukemic transformation in primary myelofibrosis oleh Valentina Poletto, Laura Villani, Paolo Catarsi, Rita Campanelli, Margherita Massa, Alessandro M. Vannucchi, Vittorio Rosti, Giovanni Barosi

    Diterbitkan 2013-08-01
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  7. 7
    In vitro megakaryocyte differentiation and proplatelet formation in Ph-negative classical myeloproliferative neoplasms: distinct patterns in the different clinical phenotypes.

    In vitro megakaryocyte differentiation and proplatelet formation in Ph-negative classical myeloproliferative neoplasms: distinct patterns in the different clinical phenotypes. oleh Alessandra Balduini, Stefania Badalucco, Maria Teresa Pugliano, Denis Baev, Annalisa De Silvestri, Marco Cattaneo, Vittorio Rosti, Giovanni Barosi

    Diterbitkan 2011-01-01
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  8. 8
    Constitutive STAT5 phosphorylation in CD34+ cells of patients with primary myelofibrosis: Correlation with driver mutation status and disease severity.

    Constitutive STAT5 phosphorylation in CD34+ cells of patients with primary myelofibrosis: Correlation with driver mutation status and disease severity. oleh Carlotta Abbà, Rita Campanelli, Paolo Catarsi, Laura Villani, Vittorio Abbonante, Melania Antonietta Sesta, Giovanni Barosi, Vittorio Rosti, Margherita Massa

    Diterbitkan 2019-01-01
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  9. 9
    JAK2 V617F genotype is a strong determinant of blast transformation in primary myelofibrosis.

    JAK2 V617F genotype is a strong determinant of blast transformation in primary myelofibrosis. oleh Giovanni Barosi, Valentina Poletto, Margherita Massa, Rita Campanelli, Laura Villani, Elisa Bonetti, Gianluca Viarengo, Paolo Catarsi, Catherine Klersy, Vittorio Rosti

    Diterbitkan 2013-01-01
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  10. 10
    JAK2 exon 14 skipping in patients with primary myelofibrosis: a minor splice variant modulated by the JAK2-V617F allele burden.

    JAK2 exon 14 skipping in patients with primary myelofibrosis: a minor splice variant modulated by the JAK2-V617F allele burden. oleh Paolo Catarsi, Vittorio Rosti, Giacomo Morreale, Valentina Poletto, Laura Villani, Roberto Bertorelli, Matteo Pedrazzini, Michele Zorzetto, Giovanni Barosi, AGIMM investigators

    Diterbitkan 2015-01-01
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  11. 11
    VEGFA rs3025039 is associated with phenotype severity of myelofibrosis‐type megakaryocyte dysplasia

    VEGFA rs3025039 is associated with phenotype severity of myelofibrosis‐type megakaryocyte dysplasia oleh Giovanni Barosi, Paolo Catarsi, Rita Campanelli, Margherita Massa, Adriana Carolei, Carlotta Abbà, Annalisa deSilvestri, Robert Peter Gale, Vittorio Rosti

    Diterbitkan 2023-08-01
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  12. 12
    A subpopulation of circulating endothelial cells express CD109 and is enriched in the blood of cancer patients.

    A subpopulation of circulating endothelial cells express CD109 and is enriched in the blood of cancer patients. oleh Patrizia Mancuso, Angelica Calleri, Giuliana Gregato, Valentina Labanca, Jessica Quarna, Pierluigi Antoniotti, Lucia Cuppini, Gaetano Finocchiaro, Marica Eoli, Vittorio Rosti, Francesco Bertolini

    Diterbitkan 2014-01-01
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  13. 13
    Shared and Distinctive Ultrastructural Abnormalities Expressed by Megakaryocytes in Bone Marrow and Spleen From Patients With Myelofibrosis

    Shared and Distinctive Ultrastructural Abnormalities Expressed by Megakaryocytes in Bone Marrow and Spleen From Patients With Myelofibrosis oleh Maria Zingariello, Vittorio Rosti, Alessandro M. Vannucchi, Paola Guglielmelli, Maria Mazzarini, Giovanni Barosi, Maria Luisa Genova, Anna Rita Migliaccio, Anna Rita Migliaccio

    Diterbitkan 2020-11-01
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  14. 14
    VEGFA rs3025020 Polymorphism Contributes to CALR-Mutation Susceptibility and Is Associated with Low Risk of Deep Vein Thrombosis in Primary Myelofibrosis

    VEGFA rs3025020 Polymorphism Contributes to CALR-Mutation Susceptibility and Is Associated with Low Risk of Deep Vein Thrombosis in Primary Myelofibrosis oleh Laura Villani, Vittorio Rosti, Margherita Massa, Rita Campanelli, Paolo Catarsi, Adriana Carolei, Carlotta Abbà, Annalisa de Silvstri, Robert Peter Gale, Giovanni Barosi

    Diterbitkan 2021-10-01
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  15. 15
    Involvement of TGFβ1 in autocrine regulation of proplatelet formation in healthy subjects and patients with primary myelofibrosis

    Involvement of TGFβ1 in autocrine regulation of proplatelet formation in healthy subjects and patients with primary myelofibrosis oleh Stefania Badalucco, Christian Andrea Di Buduo, Rita Campanelli, Isabella Pallotta, Paolo Catarsi, Vittorio Rosti, David L. Kaplan, Giovanni Barosi, Margherita Massa, Alessandra Balduini

    Diterbitkan 2013-04-01
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  16. 16
    Evidence that prefibrotic myelofibrosis is aligned along a clinical and biological continuum featuring primary myelofibrosis.

    Evidence that prefibrotic myelofibrosis is aligned along a clinical and biological continuum featuring primary myelofibrosis. oleh Giovanni Barosi, Vittorio Rosti, Elisa Bonetti, Rita Campanelli, Adriana Carolei, Paolo Catarsi, Antonina M Isgrò, Letizia Lupo, Margherita Massa, Valentina Poletto, Gianluca Viarengo, Laura Villani, Umberto Magrini

    Diterbitkan 2012-01-01
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  17. 17
    Prevalence and pathogenesis of anemia in inflammatory bowel disease. Influence of anti-tumor necrosis factor-α treatment

    Prevalence and pathogenesis of anemia in inflammatory bowel disease. Influence of anti-tumor necrosis factor-α treatment oleh Gaetano Bergamaschi, Antonio Di Sabatino, Riccardo Albertini, Sandro Ardizzone, Paolo Biancheri, Elisa Bonetti, Andrea Cassinotti, Paolo Cazzola, Konstantinos Markopoulos, Alessandro Massari, Vittorio Rosti, Gabriele Bianchi Porro, Gino R. Corazza

    Diterbitkan 2010-02-01
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  18. 18
    Congenital amegakaryocytic thrombocytopenia: clinical and biological consequences of five novel mutations

    Congenital amegakaryocytic thrombocytopenia: clinical and biological consequences of five novel mutations oleh Anna Savoia, Carlo Dufour, Franco Locatelli, Patrizia Noris, Chiara Ambaglio, Vittorio Rosti, Marco Zecca, Simona Ferrari, Filomena di Bari, Anna Corcione, Mariateresa Di Stazio, Marco Seri, Carlo L. Balduini

    Diterbitkan 2007-09-01
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  19. 19
    High frequency of endothelial colony forming cells marks a non-active myeloproliferative neoplasm with high risk of splanchnic vein thrombosis.

    High frequency of endothelial colony forming cells marks a non-active myeloproliferative neoplasm with high risk of splanchnic vein thrombosis. oleh Vittorio Rosti, Elisa Bonetti, Gaetano Bergamaschi, Rita Campanelli, Paola Guglielmelli, Marcello Maestri, Umberto Magrini, Margherita Massa, Carmine Tinelli, Gianluca Viarengo, Laura Villani, Massimo Primignani, Alessandro M Vannucchi, Francesco Frassoni, Giovanni Barosi, AGIMM Investigators

    Diterbitkan 2010-12-01
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  20. 20
    NATURAL HISTORY OF RALD: A 20 YEAR FOLLOW-UP OF A NRAS MUTATED PATIENT EXCLUDING A MALIGNANT PROGRESSION

    NATURAL HISTORY OF RALD: A 20 YEAR FOLLOW-UP OF A NRAS MUTATED PATIENT EXCLUDING A MALIGNANT PROGRESSION oleh Enrico Attardi, Beatrice Rivalta, Cristina Cifaldi, Vittorio Rosti, Lucia Pacillo, Hajro Hajrullaj, Silvia Di Cesare, Matteo Luciani, Federica Barzaghi, Andrea Finocchi, Gigliola Di Matteo, Alessandro Aiuti, Franco Locatelli, Maria Teresa Voso, Giuseppe Palumbo, Caterina Cancrini

    Diterbitkan 2023-12-01
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