Hasil Pencarian - Toshihisa Komori

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  1. 1
    Whole Aspect of Runx2 Functions in Skeletal Development

    Whole Aspect of Runx2 Functions in Skeletal Development oleh Toshihisa Komori

    Diterbitkan 2022-05-01
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  2. 2
    Regulation of Skeletal Development and Maintenance by Runx2 and Sp7

    Regulation of Skeletal Development and Maintenance by Runx2 and Sp7 oleh Toshihisa Komori

    Diterbitkan 2024-09-01
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  3. 3
    Regulation of Proliferation, Differentiation and Functions of Osteoblasts by Runx2

    Regulation of Proliferation, Differentiation and Functions of Osteoblasts by Runx2 oleh Toshihisa Komori

    Diterbitkan 2019-04-01
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  4. 4
    Molecular Processes in Chondrocyte Biology

    Molecular Processes in Chondrocyte Biology oleh Toshihisa Komori

    Diterbitkan 2020-06-01
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  5. 5
    Cell Death in Chondrocytes, Osteoblasts, and Osteocytes

    Cell Death in Chondrocytes, Osteoblasts, and Osteocytes oleh Toshihisa Komori

    Diterbitkan 2016-12-01
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  6. 6
    Functions of Osteocalcin in Bone, Pancreas, Testis, and Muscle

    Functions of Osteocalcin in Bone, Pancreas, Testis, and Muscle oleh Toshihisa Komori

    Diterbitkan 2020-10-01
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  7. 7
    Lack of reproducibility in osteocalcin-deficient mice.

    Lack of reproducibility in osteocalcin-deficient mice. oleh Takeshi Moriishi, Toshihisa Komori

    Diterbitkan 2020-06-01
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  8. 8
    Osteocytes: Their Lacunocanalicular Structure and Mechanoresponses

    Osteocytes: Their Lacunocanalicular Structure and Mechanoresponses oleh Takeshi Moriishi, Toshihisa Komori

    Diterbitkan 2022-04-01
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  9. 9
    Thrombospondin-1 Is a Putative Target Gene of Runx2 and Runx3

    Thrombospondin-1 Is a Putative Target Gene of Runx2 and Runx3 oleh Wenguang Liu, Toshihisa Komori, Xianlu Zeng, Xueqing Ba, Linghui Wang, Xiuming Shi, Vishwa Deepak

    Diterbitkan 2013-07-01
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  10. 10
    Osteocyte network; a negative regulatory system for bone mass augmented by the induction of Rankl in osteoblasts and Sost in osteocytes at unloading.

    Osteocyte network; a negative regulatory system for bone mass augmented by the induction of Rankl in osteoblasts and Sost in osteocytes at unloading. oleh Takeshi Moriishi, Ryo Fukuyama, Masako Ito, Toshihiro Miyazaki, Takafumi Maeno, Yosuke Kawai, Hisato Komori, Toshihisa Komori

    Diterbitkan 2012-01-01
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  11. 11
    Bcl2 deficiency activates FoxO through Akt inactivation and accelerates osteoblast differentiation.

    Bcl2 deficiency activates FoxO through Akt inactivation and accelerates osteoblast differentiation. oleh Takeshi Moriishi, Yosuke Kawai, Hisato Komori, Satoshi Rokutanda, Yutaka Eguchi, Yoshihide Tsujimoto, Izumi Asahina, Toshihisa Komori

    Diterbitkan 2014-01-01
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  12. 12
    Different Requirements of CBFB and RUNX2 in Skeletal Development among Calvaria, Limbs, Vertebrae and Ribs

    Different Requirements of CBFB and RUNX2 in Skeletal Development among Calvaria, Limbs, Vertebrae and Ribs oleh Qing Jiang, Xin Qin, Kenichi Nagano, Hisato Komori, Yuki Matsuo, Ichiro Taniuchi, Kosei Ito, Toshihisa Komori

    Diterbitkan 2022-10-01
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  13. 13
    <i>Bcl2l1</i> Deficiency in Osteoblasts Reduces the Trabecular Bone Due to Enhanced Osteoclastogenesis Likely through Osteoblast Apoptosis

    <i>Bcl2l1</i> Deficiency in Osteoblasts Reduces the Trabecular Bone Due to Enhanced Osteoclastogenesis Likely through Osteoblast Apoptosis oleh Takeshi Moriishi, Yosuke Kawai, Ryo Fukuyama, Yuki Matsuo, You-Wen He, Haruhiko Akiyama, Izumi Asahina, Toshihisa Komori

    Diterbitkan 2023-12-01
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  14. 14
    Expression of a Constitutively Active Form of <i>Hck</i> in Chondrocytes Activates Wnt and Hedgehog Signaling Pathways, and Induces Chondrocyte Proliferation in Mice

    Expression of a Constitutively Active Form of <i>Hck</i> in Chondrocytes Activates Wnt and Hedgehog Signaling Pathways, and Induces Chondrocyte Proliferation in Mice oleh Viviane K. S. Kawata Matsuura, Carolina Andrea Yoshida, Hisato Komori, Chiharu Sakane, Kei Yamana, Qing Jiang, Toshihisa Komori

    Diterbitkan 2020-04-01
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  15. 15
    Sp7 Transgenic Mice with a Markedly Impaired Lacunocanalicular Network Induced Sost and Reduced Bone Mass by Unloading

    Sp7 Transgenic Mice with a Markedly Impaired Lacunocanalicular Network Induced Sost and Reduced Bone Mass by Unloading oleh Takeshi Moriishi, Takuro Ito, Ryo Fukuyama, Xin Qin, Hisato Komori, Hitomi Kaneko, Yuki Matsuo, Noriaki Yoshida, Toshihisa Komori

    Diterbitkan 2022-03-01
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  16. 16
    Roles of Sp7 in osteoblasts for the proliferation, differentiation, and osteocyte process formation

    Roles of Sp7 in osteoblasts for the proliferation, differentiation, and osteocyte process formation oleh Qing Jiang, Kenichi Nagano, Takeshi Moriishi, Hisato Komori, Chiharu Sakane, Yuki Matsuo, Zhiguo Zhang, Riko Nishimura, Kosei Ito, Xin Qin, Toshihisa Komori

    Diterbitkan 2024-07-01
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  17. 17
    Antxr1, Which is a Target of Runx2, Regulates Chondrocyte Proliferation and Apoptosis

    Antxr1, Which is a Target of Runx2, Regulates Chondrocyte Proliferation and Apoptosis oleh Qing Jiang, Xin Qin, Carolina Andrea Yoshida, Hisato Komori, Kei Yamana, Shinsuke Ohba, Hironori Hojo, Brad St. Croix, Viviane K. S. Kawata-Matsuura, Toshihisa Komori

    Diterbitkan 2020-03-01
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  18. 18
    Runx2 is essential for the transdifferentiation of chondrocytes into osteoblasts.

    Runx2 is essential for the transdifferentiation of chondrocytes into osteoblasts. oleh Xin Qin, Qing Jiang, Kenichi Nagano, Takeshi Moriishi, Toshihiro Miyazaki, Hisato Komori, Kosei Ito, Klaus von der Mark, Chiharu Sakane, Hitomi Kaneko, Toshihisa Komori

    Diterbitkan 2020-11-01
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  19. 19
    Runx2 Regulates <i>Galnt3</i> and <i>Fgf23</i> Expressions and Galnt3 Decelerates Osteoid Mineralization by Stabilizing Fgf23

    Runx2 Regulates <i>Galnt3</i> and <i>Fgf23</i> Expressions and Galnt3 Decelerates Osteoid Mineralization by Stabilizing Fgf23 oleh Qing Jiang, Xin Qin, Takeshi Moriishi, Ryo Fukuyama, Shinichi Katsumata, Hiroshi Matsuzaki, Hisato Komori, Yuki Matsuo, Chiharu Sakane, Kosei Ito, Hironori Hojo, Shinsuke Ohba, Toshihisa Komori

    Diterbitkan 2024-02-01
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  20. 20
    Osteocalcin is necessary for the alignment of apatite crystallites, but not glucose metabolism, testosterone synthesis, or muscle mass.

    Osteocalcin is necessary for the alignment of apatite crystallites, but not glucose metabolism, testosterone synthesis, or muscle mass. oleh Takeshi Moriishi, Ryosuke Ozasa, Takuya Ishimoto, Takayoshi Nakano, Tomoka Hasegawa, Toshihiro Miyazaki, Wenguang Liu, Ryo Fukuyama, Yuying Wang, Hisato Komori, Xin Qin, Norio Amizuka, Toshihisa Komori

    Diterbitkan 2020-05-01
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