Hasil Pencarian - Richard Longnecker

Perbaiki Hasil
  1. 1
    The Herpes Simplex Virus Neurovirulence Factor γ34.5: Revealing Virus-Host Interactions.

    The Herpes Simplex Virus Neurovirulence Factor γ34.5: Revealing Virus-Host Interactions. oleh Douglas R Wilcox, Richard Longnecker

    Diterbitkan 2016-03-01
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  2. 2
    The Cytoplasmic Tail Domain of Epstein-Barr Virus gH Regulates Membrane Fusion Activity through Altering gH Binding to gp42 and Epithelial Cell Attachment

    The Cytoplasmic Tail Domain of Epstein-Barr Virus gH Regulates Membrane Fusion Activity through Altering gH Binding to gp42 and Epithelial Cell Attachment oleh Jia Chen, Theodore S. Jardetzky, Richard Longnecker

    Diterbitkan 2016-12-01
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  3. 3
    The Innate Immune Response to Herpes Simplex Virus 1 Infection Is Dampened in the Newborn Brain and Can Be Modulated by Exogenous Interferon Beta To Improve Survival

    The Innate Immune Response to Herpes Simplex Virus 1 Infection Is Dampened in the Newborn Brain and Can Be Modulated by Exogenous Interferon Beta To Improve Survival oleh Daniel Giraldo, Douglas R. Wilcox, Richard Longnecker

    Diterbitkan 2020-06-01
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  4. 4
    Modulation of Epstein-Barr Virus Glycoprotein B (gB) Fusion Activity by the gB Cytoplasmic Tail Domain

    Modulation of Epstein-Barr Virus Glycoprotein B (gB) Fusion Activity by the gB Cytoplasmic Tail Domain oleh Nicholas J. Garcia, Jia Chen, Richard Longnecker

    Diterbitkan 2013-03-01
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  5. 5
    Two Pathways of p27<sup>Kip1</sup> Degradation Are Required for Murine Lymphoma Driven by Myc and EBV Latent Membrane Protein 2A

    Two Pathways of p27<sup>Kip1</sup> Degradation Are Required for Murine Lymphoma Driven by Myc and EBV Latent Membrane Protein 2A oleh Richard P. Sora, Masato Ikeda, Richard Longnecker

    Diterbitkan 2019-04-01
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  6. 6
    Membrane Anchoring of Epstein-Barr Virus gp42 Inhibits Fusion with B Cells Even with Increased Flexibility Allowed by Engineered Spacers

    Membrane Anchoring of Epstein-Barr Virus gp42 Inhibits Fusion with B Cells Even with Increased Flexibility Allowed by Engineered Spacers oleh Cynthia L. Rowe, Jia Chen, Theodore S. Jardetzky, Richard Longnecker

    Diterbitkan 2015-02-01
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  7. 7
    The KGD Motif of Epstein-Barr Virus gH/gL Is Bifunctional, Orchestrating Infection of B Cells and Epithelial Cells

    The KGD Motif of Epstein-Barr Virus gH/gL Is Bifunctional, Orchestrating Infection of B Cells and Epithelial Cells oleh Jia Chen, Cynthia L. Rowe, Theodore S. Jardetzky, Richard Longnecker

    Diterbitkan 2012-03-01
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  8. 8
    The Type I Interferon Response Determines Differences in Choroid Plexus Susceptibility between Newborns and Adults in Herpes Simplex Virus Encephalitis

    The Type I Interferon Response Determines Differences in Choroid Plexus Susceptibility between Newborns and Adults in Herpes Simplex Virus Encephalitis oleh Douglas R. Wilcox, Stephen S. Folmsbee, William J. Muller, Richard Longnecker

    Diterbitkan 2016-05-01
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  9. 9
    Epstein-Barr virus LMP2A reduces hyperactivation induced by LMP1 to restore normal B cell phenotype in transgenic mice.

    Epstein-Barr virus LMP2A reduces hyperactivation induced by LMP1 to restore normal B cell phenotype in transgenic mice. oleh Alexandra C Vrazo, Maria Chauchard, Nancy Raab-Traub, Richard Longnecker

    Diterbitkan 2012-01-01
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  10. 10
    Structure-Based Mutations in the Herpes Simplex Virus 1 Glycoprotein B Ectodomain Arm Impart a Slow-Entry Phenotype

    Structure-Based Mutations in the Herpes Simplex Virus 1 Glycoprotein B Ectodomain Arm Impart a Slow-Entry Phenotype oleh Qing Fan, Sarah J. Kopp, Sarah A. Connolly, Richard Longnecker

    Diterbitkan 2017-07-01
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  11. 11
    Ephrin Receptor A4 is a New Kaposi’s Sarcoma-Associated Herpesvirus Virus Entry Receptor

    Ephrin Receptor A4 is a New Kaposi’s Sarcoma-Associated Herpesvirus Virus Entry Receptor oleh Jia Chen, Xianming Zhang, Samantha Schaller, Theodore S. Jardetzky, Richard Longnecker

    Diterbitkan 2019-02-01
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  12. 12
    Erratum for Riccio et al., “Characterization of Sex Differences in Ocular Herpes Simplex Virus 1 Infection and Herpes Stromal Keratitis Pathogenesis of Wild-Type and Herpesvirus Entry Mediator Knockout Mice”

    Erratum for Riccio et al., “Characterization of Sex Differences in Ocular Herpes Simplex Virus 1 Infection and Herpes Stromal Keratitis Pathogenesis of Wild-Type and Herpesvirus En... oleh Rachel E. Riccio, Seo J. Park, Richard Longnecker, Sarah J. Kopp

    Diterbitkan 2019-06-01
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  13. 13
    Differential reliance on autophagy for protection from HSV encephalitis between newborns and adults.

    Differential reliance on autophagy for protection from HSV encephalitis between newborns and adults. oleh Douglas R Wilcox, Nitin R Wadhwani, Richard Longnecker, William J Muller

    Diterbitkan 2015-01-01
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  14. 14
    Characterization of Sex Differences in Ocular Herpes Simplex Virus 1 Infection and Herpes Stromal Keratitis Pathogenesis of Wild-Type and Herpesvirus Entry Mediator Knockout Mice

    Characterization of Sex Differences in Ocular Herpes Simplex Virus 1 Infection and Herpes Stromal Keratitis Pathogenesis of Wild-Type and Herpesvirus Entry Mediator Knockout Mice oleh Rachel E. Riccio, Seo J. Park, Richard Longnecker, Sarah J. Kopp

    Diterbitkan 2019-04-01
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  15. 15
    Multiple Sites on Glycoprotein H (gH) Functionally Interact with the gB Fusion Protein to Promote Fusion during Herpes Simplex Virus (HSV) Entry

    Multiple Sites on Glycoprotein H (gH) Functionally Interact with the gB Fusion Protein to Promote Fusion during Herpes Simplex Virus (HSV) Entry oleh Qing Fan, Daniel P. Hippler, Yueqi Yang, Richard Longnecker, Sarah A. Connolly

    Diterbitkan 2023-02-01
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  16. 16
    Structural basis for Epstein–Barr virus host cell tropism mediated by gp42 and gHgL entry glycoproteins

    Structural basis for Epstein–Barr virus host cell tropism mediated by gp42 and gHgL entry glycoproteins oleh Karthik Sathiyamoorthy, Yao Xiong Hu, Britta S. Möhl, Jia Chen, Richard Longnecker, Theodore S. Jardetzky

    Diterbitkan 2016-12-01
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  17. 17
    Natural Selection of Glycoprotein B Mutations That Rescue the Small-Plaque Phenotype of a Fusion-Impaired Herpes Simplex Virus Mutant

    Natural Selection of Glycoprotein B Mutations That Rescue the Small-Plaque Phenotype of a Fusion-Impaired Herpes Simplex Virus Mutant oleh Qing Fan, Sarah J. Kopp, Nina C. Byskosh, Sarah A. Connolly, Richard Longnecker

    Diterbitkan 2018-11-01
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  18. 18
    Herpesvirus Entry Mediator on Radiation-Resistant Cell Lineages Promotes Ocular Herpes Simplex Virus 1 Pathogenesis in an Entry-Independent Manner

    Herpesvirus Entry Mediator on Radiation-Resistant Cell Lineages Promotes Ocular Herpes Simplex Virus 1 Pathogenesis in an Entry-Independent Manner oleh Rebecca G. Edwards, Sarah J. Kopp, Andrew H. Karaba, Douglas R. Wilcox, Richard Longnecker

    Diterbitkan 2015-10-01
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  19. 19
    Herpesvirus Entry Mediator Binding Partners Mediate Immunopathogenesis of Ocular Herpes Simplex Virus 1 Infection

    Herpesvirus Entry Mediator Binding Partners Mediate Immunopathogenesis of Ocular Herpes Simplex Virus 1 Infection oleh Seo J. Park, Rachel E. Riccio, Sarah J. Kopp, Igal Ifergan, Stephen D. Miller, Richard Longnecker

    Diterbitkan 2020-06-01
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  20. 20
    ASC-dependent inflammasomes contribute to immunopathology and mortality in herpes simplex encephalitis.

    ASC-dependent inflammasomes contribute to immunopathology and mortality in herpes simplex encephalitis. oleh Cooper K Hayes, Douglas R Wilcox, Yuchen Yang, Grace K Coleman, Melissa A Brown, Richard Longnecker

    Diterbitkan 2021-02-01
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