Hasil Pencarian - Raymond P Boot-Handford

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  1. 1
    Increased classical endoplasmic reticulum stress is sufficient to reduce chondrocyte proliferation rate in the growth plate and decrease bone growth.

    Increased classical endoplasmic reticulum stress is sufficient to reduce chondrocyte proliferation rate in the growth plate and decrease bone growth. oleh Louise H W Kung, M Helen Rajpar, Richard Preziosi, Michael D Briggs, Raymond P Boot-Handford

    Diterbitkan 2015-01-01
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  2. 2
    A novel transgenic mouse model of growth plate dysplasia reveals that decreased chondrocyte proliferation due to chronic ER stress is a key factor in reduced bone growth

    A novel transgenic mouse model of growth plate dysplasia reveals that decreased chondrocyte proliferation due to chronic ER stress is a key factor in reduced bone growth oleh Benedetta Gualeni, M. Helen Rajpar, Aaron Kellogg, Peter A. Bell, Peter Arvan, Raymond P. Boot-Handford, Michael D. Briggs

    Diterbitkan 2013-11-01
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  3. 3
    Abnormal chondrocyte apoptosis in the cartilage growth plate is influenced by genetic background and deletion of CHOP in a targeted mouse model of pseudoachondroplasia.

    Abnormal chondrocyte apoptosis in the cartilage growth plate is influenced by genetic background and deletion of CHOP in a targeted mouse model of pseudoachondroplasia. oleh Katarzyna A Piróg, Andreja Irman, Siobhan Young, Poonam Halai, Peter A Bell, Raymond P Boot-Handford, Michael D Briggs

    Diterbitkan 2014-01-01
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  4. 4
    Collagen XXVII organises the pericellular matrix in the growth plate.

    Collagen XXVII organises the pericellular matrix in the growth plate. oleh Darren A Plumb, Laila Ferrara, Tanja Torbica, Lynnette Knowles, Aleksandr Mironov, Karl E Kadler, Michael D Briggs, Raymond P Boot-Handford

    Diterbitkan 2011-01-01
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  5. 5
    Cartilage endoplasmic reticulum stress may influence the onset but not the progression of experimental osteoarthritis

    Cartilage endoplasmic reticulum stress may influence the onset but not the progression of experimental osteoarthritis oleh Louise H. W. Kung, Lorna Mullan, Jamie Soul, Ping Wang, Kazutoshi Mori, John F. Bateman, Michael D. Briggs, Raymond P. Boot-Handford

    Diterbitkan 2019-09-01
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  6. 6
    Arhgap28 is a RhoGAP that inactivates RhoA and downregulates stress fibers.

    Arhgap28 is a RhoGAP that inactivates RhoA and downregulates stress fibers. oleh Ching-Yan Chloé Yeung, Susan H Taylor, Richa Garva, David F Holmes, Leo A Zeef, Raija Soininen, Raymond P Boot-Handford, Karl E Kadler

    Diterbitkan 2014-01-01
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  7. 7
    Transcriptional profiling of chondrodysplasia growth plate cartilage reveals adaptive ER-stress networks that allow survival but disrupt hypertrophy.

    Transcriptional profiling of chondrodysplasia growth plate cartilage reveals adaptive ER-stress networks that allow survival but disrupt hypertrophy. oleh Trevor L Cameron, Katrina M Bell, Liliana Tatarczuch, Eleanor J Mackie, M Helen Rajpar, Ben T McDermott, Raymond P Boot-Handford, John F Bateman

    Diterbitkan 2011-01-01
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  8. 8
    XBP1-Independent UPR Pathways Suppress C/EBP-β Mediated Chondrocyte Differentiation in ER-Stress Related Skeletal Disease.

    XBP1-Independent UPR Pathways Suppress C/EBP-β Mediated Chondrocyte Differentiation in ER-Stress Related Skeletal Disease. oleh Trevor L Cameron, Katrina M Bell, Irma L Gresshoff, Lisa Sampurno, Lorna Mullan, Joerg Ermann, Laurie H Glimcher, Raymond P Boot-Handford, John F Bateman

    Diterbitkan 2015-09-01
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  9. 9
    Analysis of the cartilage proteome from three different mouse models of genetic skeletal diseases reveals common and discrete disease signatures

    Analysis of the cartilage proteome from three different mouse models of genetic skeletal diseases reveals common and discrete disease signatures oleh Peter A. Bell, Raimund Wagener, Frank Zaucke, Manuel Koch, Julian Selley, Stacey Warwood, David Knight, Raymond P. Boot-Handford, David J. Thornton, Michael D. Briggs

    Diterbitkan 2013-06-01
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  10. 10
    XBP1 signalling is essential for alleviating mutant protein aggregation in ER-stress related skeletal disease.

    XBP1 signalling is essential for alleviating mutant protein aggregation in ER-stress related skeletal disease. oleh Katarzyna A Piróg, Ella P Dennis, Claire L Hartley, Robert M Jackson, Jamie Soul, Jean-Marc Schwartz, John F Bateman, Raymond P Boot-Handford, Michael D Briggs

    Diterbitkan 2019-07-01
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  11. 11
    Targeted induction of endoplasmic reticulum stress induces cartilage pathology.

    Targeted induction of endoplasmic reticulum stress induces cartilage pathology. oleh M Helen Rajpar, Ben McDermott, Louise Kung, Rachel Eardley, Lynette Knowles, Mel Heeran, David J Thornton, Richard Wilson, John F Bateman, Richard Poulsom, Peter Arvan, Karl E Kadler, Michael D Briggs, Raymond P Boot-Handford

    Diterbitkan 2009-10-01
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