Hasil Pencarian - Ranjithkumar Rajendran

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  1. 1
    FGF/FGFR Pathways in Multiple Sclerosis and in Its Disease Models

    FGF/FGFR Pathways in Multiple Sclerosis and in Its Disease Models oleh Ranjithkumar Rajendran, Gregor Böttiger, Christine Stadelmann, Srikanth Karnati, Martin Berghoff

    Diterbitkan 2021-04-01
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  2. 2
    Interferon Beta-1a versus Combined Interferon Beta-1a and Oligodendrocyte-Specific FGFR1 Deletion in Experimental Autoimmune Encephalomyelitis

    Interferon Beta-1a versus Combined Interferon Beta-1a and Oligodendrocyte-Specific FGFR1 Deletion in Experimental Autoimmune Encephalomyelitis oleh Ranjithkumar Rajendran, Vinothkumar Rajendran, Liza Gupta, Kian Shirvanchi, Darja Schunin, Srikanth Karnati, Mario Giraldo-Velásquez, Martin Berghoff

    Diterbitkan 2022-10-01
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  3. 3
    Effects of FGFR Tyrosine Kinase Inhibition in OLN-93 Oligodendrocytes

    Effects of FGFR Tyrosine Kinase Inhibition in OLN-93 Oligodendrocytes oleh Ranjithkumar Rajendran, Gregor Böttiger, Niklas Dentzien, Vinothkumar Rajendran, Bischand Sharifi, Süleyman Ergün, Christine Stadelmann, Srikanth Karnati, Martin Berghoff

    Diterbitkan 2021-05-01
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  4. 4
    Oligodendrocyte-Specific Deletion of <i>FGFR1</i> Reduces Cerebellar Inflammation and Neurodegeneration in MOG<sub>35-55</sub>-Induced EAE

    Oligodendrocyte-Specific Deletion of <i>FGFR1</i> Reduces Cerebellar Inflammation and Neurodegeneration in MOG<sub>35-55</sub>-Induced EAE oleh Ranjithkumar Rajendran, Vinothkumar Rajendran, Mario Giraldo-Velasquez, Fevronia-Foivi Megalofonou, Fynn Gurski, Christine Stadelmann, Srikanth Karnati, Martin Berghoff

    Diterbitkan 2021-08-01
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  5. 5
    Quantitative Lipidomic Analysis of Takotsubo Syndrome Patients' Serum

    Quantitative Lipidomic Analysis of Takotsubo Syndrome Patients' Serum oleh Srikanth Karnati, Gulcan Guntas, Ranjithkumar Rajendran, Sergey Shityakov, Marcus Höring, Gerhard Liebisch, Djuro Kosanovic, Süleyman Ergün, Michiaki Nagai, Carola Y. Förster

    Diterbitkan 2022-04-01
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  6. 6
    Tafazzin deficiency causes substantial remodeling in the lipidome of a mouse model of Barth Syndrome cardiomyopathy

    Tafazzin deficiency causes substantial remodeling in the lipidome of a mouse model of Barth Syndrome cardiomyopathy oleh Malte Hachmann, Güntas Gülcan, Ranjithkumar Rajendran, Marcus Höring, Gerhard Liebisch, Akash Bachhuka, Michael Kohlhaas, Christoph Maack, Christoph Maack, Süleyman Ergün, Jan Dudek, Srikanth Karnati

    Diterbitkan 2024-04-01
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