Hasil Pencarian - Rachel Wevrick

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  1. 1
    Loss of magel2, a candidate gene for features of Prader-Willi syndrome, impairs reproductive function in mice.

    Loss of magel2, a candidate gene for features of Prader-Willi syndrome, impairs reproductive function in mice. oleh Rebecca E Mercer, Rachel Wevrick

    Diterbitkan 2009-01-01
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  2. 2
    Preclinical Testing in Translational Animal Models of Prader-Willi Syndrome: Overview and Gap Analysis

    Preclinical Testing in Translational Animal Models of Prader-Willi Syndrome: Overview and Gap Analysis oleh K. Vanessa Carias, Rachel Wevrick

    Diterbitkan 2019-06-01
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  3. 3
    A MAGEL2-deubiquitinase complex modulates the ubiquitination of circadian rhythm protein CRY1.

    A MAGEL2-deubiquitinase complex modulates the ubiquitination of circadian rhythm protein CRY1. oleh K Vanessa Carias, Mercedes Zoeteman, Abigail Seewald, Matthea R Sanderson, Jocelyn M Bischof, Rachel Wevrick

    Diterbitkan 2020-01-01
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  4. 4
    Magel2 is required for leptin-mediated depolarization of POMC neurons in the hypothalamic arcuate nucleus in mice.

    Magel2 is required for leptin-mediated depolarization of POMC neurons in the hypothalamic arcuate nucleus in mice. oleh Rebecca E Mercer, Sheldon D Michaelson, Melissa J S Chee, Tanya A Atallah, Rachel Wevrick, William F Colmers

    Diterbitkan 2013-01-01
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  5. 5
    Hypothalamic AAV-BDNF gene therapy improves metabolic function and behavior in the Magel2-null mouse model of Prader-Willi syndrome

    Hypothalamic AAV-BDNF gene therapy improves metabolic function and behavior in the Magel2-null mouse model of Prader-Willi syndrome oleh Nicholas J. Queen, Xunchang Zou, Jacqueline M. Anderson, Wei Huang, Bhavya Appana, Suraj Komatineni, Rachel Wevrick, Lei Cao

    Diterbitkan 2022-12-01
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  6. 6
    The Smc5/Smc6/MAGE complex confers resistance to caffeine and genotoxic stress in Drosophila melanogaster.

    The Smc5/Smc6/MAGE complex confers resistance to caffeine and genotoxic stress in Drosophila melanogaster. oleh Xiao Li, Ran Zhuo, Stanley Tiong, Francesca Di Cara, Kirst King-Jones, Sarah C Hughes, Shelagh D Campbell, Rachel Wevrick

    Diterbitkan 2013-01-01
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  7. 7
    ROHHAD and Prader-Willi syndrome (PWS): clinical and genetic comparison

    ROHHAD and Prader-Willi syndrome (PWS): clinical and genetic comparison oleh Sarah F. Barclay, Casey M. Rand, Lisa Nguyen, Richard J. A. Wilson, Rachel Wevrick, William T. Gibson, N. Torben Bech-Hansen, Debra E. Weese-Mayer

    Diterbitkan 2018-07-01
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  8. 8
    Stochastic loss of silencing of the imprinted Ndn/NDN allele, in a mouse model and humans with prader-willi syndrome, has functional consequences.

    Stochastic loss of silencing of the imprinted Ndn/NDN allele, in a mouse model and humans with prader-willi syndrome, has functional consequences. oleh Anne Rieusset, Fabienne Schaller, Unga Unmehopa, Valery Matarazzo, Françoise Watrin, Matthias Linke, Beatrice Georges, Jocelyn Bischof, Femke Dijkstra, Monique Bloemsma, Severine Corby, François J Michel, Rachel Wevrick, Ulrich Zechner, Dick Swaab, Keith Dudley, Laurent Bezin, Françoise Muscatelli

    Diterbitkan 2013-01-01
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  9. 9
    Targeting the endocannabinoid/CB1 receptor system for treating obesity in Prader–Willi syndrome

    Targeting the endocannabinoid/CB1 receptor system for treating obesity in Prader–Willi syndrome oleh Ibrahim Knani, Brian J. Earley, Shiran Udi, Alina Nemirovski, Rivka Hadar, Asaad Gammal, Resat Cinar, Harry J. Hirsch, Yehuda Pollak, Itai Gross, Talia Eldar-Geva, Daniela P. Reyes-Capo, Joan C. Han, Andrea M. Haqq, Varda Gross-Tsur, Rachel Wevrick, Joseph Tam

    Diterbitkan 2016-12-01
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