Hasil Pencarian - Patrice X. Petit

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  1. 1
    Sodium arsenite and arsenic trioxide differently affect the oxidative stress of lymphoblastoid cells: An intricate crosstalk between mitochondria, autophagy and cell death

    Sodium arsenite and arsenic trioxide differently affect the oxidative stress of lymphoblastoid cells: An intricate crosstalk between mitochondria, autophagy and cell death oleh Nathan Earl Rainey, Anne-Sophie Armand, Patrice X. Petit

    Diterbitkan 2024-01-01
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  2. 2
    Tafazzin Mutation Affecting Cardiolipin Leads to Increased Mitochondrial Superoxide Anions and Mitophagy Inhibition in Barth Syndrome

    Tafazzin Mutation Affecting Cardiolipin Leads to Increased Mitochondrial Superoxide Anions and Mitophagy Inhibition in Barth Syndrome oleh Patrice X. Petit, Hector Ardilla-Osorio, Lucile Penalvia, Nathan E. Rainey

    Diterbitkan 2020-10-01
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  3. 3
    Cross-talk between oxysterols and glucocorticoids: differential regulation of secreted phopholipase A2 and impact on oligodendrocyte death.

    Cross-talk between oxysterols and glucocorticoids: differential regulation of secreted phopholipase A2 and impact on oligodendrocyte death. oleh Amalia Trousson, Joelle Makoukji, Patrice X Petit, Sophie Bernard, Christian Slomianny, Michael Schumacher, Charbel Massaad

    Diterbitkan 2009-11-01
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  4. 4
    Synergistic cellular effects including mitochondrial destabilization, autophagy and apoptosis following low-level exposure to a mixture of lipophilic persistent organic pollutants

    Synergistic cellular effects including mitochondrial destabilization, autophagy and apoptosis following low-level exposure to a mixture of lipophilic persistent organic pollutants oleh Nathan E. Rainey, Ana Saric, Alexandre Leberre, Etienne Dewailly, Christian Slomianny, Guillaume Vial, Harold I. Zeliger, Patrice X. Petit

    Diterbitkan 2017-07-01
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  5. 5
    Barth Syndrome:From mitochondrial dysfunctions associated with aberrant production of reactive oxygen species to pluripotent stem cell studies

    Barth Syndrome:From mitochondrial dysfunctions associated with aberrant production of reactive oxygen species to pluripotent stem cell studies oleh Ana eSaric, Ana eSaric, Karine eAndreau, Anne-Sophie eArmand, Ian Max Möller, Patrice X. PETIT

    Diterbitkan 2016-01-01
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  6. 6
    Mechanistic issues of the interaction of the hairpin-forming domain of tBid with mitochondrial cardiolipin.

    Mechanistic issues of the interaction of the hairpin-forming domain of tBid with mitochondrial cardiolipin. oleh François Gonzalvez, Fabrizio Pariselli, Olivier Jalmar, Pauline Dupaigne, Franck Sureau, Marc Dellinger, Eric A Hendrickson, Sophie Bernard, Patrice X Petit

    Diterbitkan 2010-01-01
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  7. 7
    Highlighting Curcumin-Induced Crosstalk between Autophagy and Apoptosis as Supported by Its Specific Subcellular Localization

    Highlighting Curcumin-Induced Crosstalk between Autophagy and Apoptosis as Supported by Its Specific Subcellular Localization oleh Francisco J. Sala de Oyanguren, Nathan E. Rainey, Aoula Moustapha, Ana Saric, Franck Sureau, José-Enrique O’Connor, Patrice X. Petit

    Diterbitkan 2020-02-01
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  8. 8
    Caspase-8 binding to cardiolipin in giant unilamellar vesicles provides a functional docking platform for bid.

    Caspase-8 binding to cardiolipin in giant unilamellar vesicles provides a functional docking platform for bid. oleh Olivier Jalmar, Liberty François-Moutal, Ana-Jesus García-Sáez, Mark Perry, Thierry Granjon, François Gonzalvez, Eyal Gottlieb, Jesus Ayala-Sanmartin, Beate Klösgen, Petra Schwille, Patrice X Petit

    Diterbitkan 2013-01-01
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  9. 9
    Decreasing cytosolic translation is beneficial to yeast and human Tafazzin-deficient cells

    Decreasing cytosolic translation is beneficial to yeast and human Tafazzin-deficient cells oleh Maxence de Taffin de Tilques, Jean-Paul Lasserre, François Godard, Elodie Sardin, Marine Bouhier, Marina Le Guedard, Roza Kucharczyk, Patrice X. Petit, Eric Testet, Jean-Paul di Rago, Déborah Tribouillard-Tanvier

    Diterbitkan 2018-02-01
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  10. 10
    COUP-TFII controls mouse pancreatic β-cell mass through GLP-1-β-catenin signaling pathways.

    COUP-TFII controls mouse pancreatic β-cell mass through GLP-1-β-catenin signaling pathways. oleh Marie Boutant, Oscar Henrique Pereira Ramos, Cécile Tourrel-Cuzin, Jamileh Movassat, Anissa Ilias, David Vallois, Julien Planchais, Jean-Paul Pégorier, Frans Schuit, Patrice X Petit, Pascale Bossard, Kathrin Maedler, Anne Grapin-Botton, Mireille Vasseur-Cognet

    Diterbitkan 2012-01-01
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