Hasil Pencarian - Pascual Sanz

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  1. 1
    Reactive Glia Inflammatory Signaling Pathways and Epilepsy

    Reactive Glia Inflammatory Signaling Pathways and Epilepsy oleh Pascual Sanz, Maria Adelaida Garcia-Gimeno

    Diterbitkan 2020-06-01
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  2. 2
    Beneficial Effects of Metformin on the Central Nervous System, with a Focus on Epilepsy and Lafora Disease

    Beneficial Effects of Metformin on the Central Nervous System, with a Focus on Epilepsy and Lafora Disease oleh Pascual Sanz, José Maria Serratosa, Marina P. Sánchez

    Diterbitkan 2021-05-01
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  3. 3
    Neuroinflammation and Epilepsy: From Pathophysiology to Therapies Based on Repurposing Drugs

    Neuroinflammation and Epilepsy: From Pathophysiology to Therapies Based on Repurposing Drugs oleh Pascual Sanz, Teresa Rubio, Maria Adelaida Garcia-Gimeno

    Diterbitkan 2024-04-01
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  4. 4
    Lafora Disease: A Ubiquitination-Related Pathology

    Lafora Disease: A Ubiquitination-Related Pathology oleh Maria Adelaida García-Gimeno, Erwin Knecht, Pascual Sanz

    Diterbitkan 2018-07-01
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  5. 5
    Structure-Function Analysis of PPP1R3D, a Protein Phosphatase 1 Targeting Subunit, Reveals a Binding Motif for 14-3-3 Proteins which Regulates its Glycogenic Properties.

    Structure-Function Analysis of PPP1R3D, a Protein Phosphatase 1 Targeting Subunit, Reveals a Binding Motif for 14-3-3 Proteins which Regulates its Glycogenic Properties. oleh Carla Rubio-Villena, Pascual Sanz, Maria Adelaida Garcia-Gimeno

    Diterbitkan 2015-01-01
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  6. 6
    Increased endoplasmic reticulum stress and decreased proteasomal function in lafora disease models lacking the phosphatase laforin.

    Increased endoplasmic reticulum stress and decreased proteasomal function in lafora disease models lacking the phosphatase laforin. oleh Santiago Vernia, Teresa Rubio, Miguel Heredia, Santiago Rodríguez de Córdoba, Pascual Sanz

    Diterbitkan 2009-06-01
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  7. 7
    TNF and IL6/Jak2 signaling pathways are the main contributors of the glia-derived neuroinflammation present in Lafora disease, a fatal form of progressive myoclonus epilepsy

    TNF and IL6/Jak2 signaling pathways are the main contributors of the glia-derived neuroinflammation present in Lafora disease, a fatal form of progressive myoclonus epilepsy oleh Teresa Rubio, Rosa Viana, Mireia Moreno-Estellés, Ángela Campos-Rodríguez, Pascual Sanz

    Diterbitkan 2023-01-01
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  8. 8
    TRIM32 and Malin in Neurological and Neuromuscular Rare Diseases

    TRIM32 and Malin in Neurological and Neuromuscular Rare Diseases oleh Lorena Kumarasinghe, Lu Xiong, Maria Adelaida Garcia-Gimeno, Elisa Lazzari, Pascual Sanz, Germana Meroni

    Diterbitkan 2021-04-01
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  9. 9
    Autophagy drives the conversion of developmental neural stem cells to the adult quiescent state

    Autophagy drives the conversion of developmental neural stem cells to the adult quiescent state oleh Isabel Calatayud-Baselga, Lucía Casares-Crespo, Carmina Franch-Ibáñez, José Guijarro-Nuez, Pascual Sanz, Helena Mira

    Diterbitkan 2023-11-01
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  10. 10
    Deciphering the Polyglucosan Accumulation Present in Lafora Disease Using an Astrocytic Cellular Model

    Deciphering the Polyglucosan Accumulation Present in Lafora Disease Using an Astrocytic Cellular Model oleh Mireia Moreno-Estellés, Ángela Campos-Rodríguez, Carla Rubio-Villena, Lorena Kumarasinghe, Maria Adelaida Garcia-Gimeno, Pascual Sanz

    Diterbitkan 2023-03-01
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  11. 11
    Dimerization of the glucan phosphatase laforin requires the participation of cysteine 329.

    Dimerization of the glucan phosphatase laforin requires the participation of cysteine 329. oleh Pablo Sánchez-Martín, Madushi Raththagala, Travis M Bridges, Satrio Husodo, Matthew S Gentry, Pascual Sanz, Carlos Romá-Mateo

    Diterbitkan 2013-01-01
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  12. 12
    Laforin, a dual specificity phosphatase involved in Lafora disease, is present mainly as monomeric form with full phosphatase activity.

    Laforin, a dual specificity phosphatase involved in Lafora disease, is present mainly as monomeric form with full phosphatase activity. oleh Vikas V Dukhande, Devin M Rogers, Carlos Romá-Mateo, Jordi Donderis, Alberto Marina, Adam O Taylor, Pascual Sanz, Matthew S Gentry

    Diterbitkan 2011-01-01
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  13. 13
    Metformin treatment reduces motor and neuropsychiatric phenotypes in the zQ175 mouse model of Huntington disease

    Metformin treatment reduces motor and neuropsychiatric phenotypes in the zQ175 mouse model of Huntington disease oleh Ana Sanchis, María Adelaida García-Gimeno, Antonio José Cañada-Martínez, María Dolores Sequedo, José María Millán, Pascual Sanz, Rafael P. Vázquez-Manrique

    Diterbitkan 2019-06-01
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  14. 14
    Gene expression analysis method integration and co-expression module detection applied to rare glucide metabolism disorders using ExpHunterSuite

    Gene expression analysis method integration and co-expression module detection applied to rare glucide metabolism disorders using ExpHunterSuite oleh Fernando M. Jabato, José Córdoba-Caballero, Elena Rojano, Carlos Romá-Mateo, Pascual Sanz, Belén Pérez, Diana Gallego, Pedro Seoane, Juan A. G. Ranea, James R. Perkins

    Diterbitkan 2021-07-01
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  15. 15
    Age-Related microRNA Overexpression in Lafora Disease Male Mice Provides Links between Neuroinflammation and Oxidative Stress

    Age-Related microRNA Overexpression in Lafora Disease Male Mice Provides Links between Neuroinflammation and Oxidative Stress oleh Carlos Romá-Mateo, Sheila Lorente-Pozo, Lucía Márquez-Thibaut, Mireia Moreno-Estellés, Concepción Garcés, Daymé González, Marcos Lahuerta, Carmen Aguado, José Luis García-Giménez, Pascual Sanz, Federico V. Pallardó

    Diterbitkan 2023-01-01
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  16. 16
    A PTG variant contributes to a milder phenotype in Lafora disease.

    A PTG variant contributes to a milder phenotype in Lafora disease. oleh Rosa Guerrero, Santiago Vernia, Raúl Sanz, Irene Abreu-Rodríguez, Carmen Almaraz, María García-Hoyos, Roberto Michelucci, Carlo Alberto Tassinari, Patrizia Riguzzi, Carlo Nobile, Pascual Sanz, José M Serratosa, Pilar Gómez-Garre

    Diterbitkan 2011-01-01
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  17. 17
    Studying Closed Hydrodynamic Models of "In Vivo" DNA Perfusion in Pig Liver for Gene Therapy Translation to Humans.

    Studying Closed Hydrodynamic Models of "In Vivo" DNA Perfusion in Pig Liver for Gene Therapy Translation to Humans. oleh Luis Sendra, Antonio Miguel, Daniel Pérez-Enguix, María José Herrero, Eva Montalvá, María Adelaida García-Gimeno, Inmaculada Noguera, Ana Díaz, Judith Pérez, Pascual Sanz, Rafael López-Andújar, Luis Martí-Bonmatí, Salvador F Aliño

    Diterbitkan 2016-01-01
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  18. 18
    Oxidative Stress, a Crossroad Between Rare Diseases and Neurodegeneration

    Oxidative Stress, a Crossroad Between Rare Diseases and Neurodegeneration oleh Carmen Espinós, Máximo Ibo Galindo, María Adelaida García-Gimeno, José Santiago Ibáñez-Cabellos, Dolores Martínez-Rubio, José María Millán, Regina Rodrigo, Pascual Sanz, Marta Seco-Cervera, Teresa Sevilla, Andrea Tapia, Federico V. Pallardó

    Diterbitkan 2020-04-01
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  19. 19
    An empirical pipeline for personalized diagnosis of Lafora disease mutations

    An empirical pipeline for personalized diagnosis of Lafora disease mutations oleh M. Kathryn Brewer, Maria Machio-Castello, Rosa Viana, Jeremiah L. Wayne, Andrea Kuchtová, Zoe R. Simmons, Sarah Sternbach, Sheng Li, Maria Adelaida García-Gimeno, Jose M. Serratosa, Pascual Sanz, Craig W. Vander Kooi, Matthew S. Gentry

    Diterbitkan 2021-11-01
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