Hasil Pencarian - Miranda D Grounds

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  1. 1
    Article Commentary: Commentary on the Present State of Knowledge for Myoblast Transfer Therapy

    Article Commentary: Commentary on the Present State of Knowledge for Myoblast Transfer Therapy oleh Miranda D. Grounds

    Diterbitkan 1996-05-01
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  2. 2
    Myoblast Transfer Therapy in the New Millennium

    Myoblast Transfer Therapy in the New Millennium oleh Miranda D. Grounds

    Diterbitkan 2000-07-01
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  3. 3
    Cromolyn administration (to block mast cell degranulation) reduces necrosis of dystrophic muscle in mdx mice

    Cromolyn administration (to block mast cell degranulation) reduces necrosis of dystrophic muscle in mdx mice oleh Hannah G. Radley, Miranda D. Grounds

    Diterbitkan 2006-08-01
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  4. 4
    Exposure to Tissue Culture Conditions Can Adversely Affect Myoblast Behavior in Vivo in Whole Muscle Grafts: Implications for Myoblast Transfer Therapy

    Exposure to Tissue Culture Conditions Can Adversely Affect Myoblast Behavior in Vivo in Whole Muscle Grafts: Implications for Myoblast Transfer Therapy oleh Gayle M. Smythe, Miranda D. Grounds

    Diterbitkan 2000-05-01
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  5. 5
    A Potential Alternative Strategy for Myoblast Transfer Therapy: The use of Sliced Muscle Grafts

    A Potential Alternative Strategy for Myoblast Transfer Therapy: The use of Sliced Muscle Grafts oleh Ying Fan, Manfred W. Beilharz, Miranda D. Grounds

    Diterbitkan 1996-05-01
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  6. 6
    Cilia, Centrosomes and Skeletal Muscle

    Cilia, Centrosomes and Skeletal Muscle oleh Dominic C. H. Ng, Uda Y. Ho, Miranda D. Grounds

    Diterbitkan 2021-09-01
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  7. 7
    Why Do Cultured Transplanted Myoblasts Die in Vivo? DNA Quantification Shows Enhanced Survival of Donor Male Myoblasts in Host Mice Depleted of CD4 and CD8 Cells or NK1.1 Cells

    Why Do Cultured Transplanted Myoblasts Die in Vivo? DNA Quantification Shows Enhanced Survival of Donor Male Myoblasts in Host Mice Depleted of CD4 and CD8 Cells or NK1.1 Cells oleh Stuart I. Hodgetts, Manfred W. Beilharz, Anthony A. Scalzo, Miranda D. Grounds

    Diterbitkan 2000-07-01
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  8. 8
    Dysferlin Deficiency Results in Myofiber-Type Specific Differences in Abundances of Calcium-Handling and Glycogen Metabolism Proteins

    Dysferlin Deficiency Results in Myofiber-Type Specific Differences in Abundances of Calcium-Handling and Glycogen Metabolism Proteins oleh Erin M. Lloyd, Gavin J. Pinniger, Miranda D. Grounds, Robyn M. Murphy

    Diterbitkan 2022-12-01
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  9. 9
    Striking denervation of neuromuscular junctions without lumbar motoneuron loss in geriatric mouse muscle.

    Striking denervation of neuromuscular junctions without lumbar motoneuron loss in geriatric mouse muscle. oleh Ruth Jinfen Chai, Jana Vukovic, Sarah Dunlop, Miranda D Grounds, Thea Shavlakadze

    Diterbitkan 2011-01-01
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  10. 10
    A Blood Biomarker for Duchenne Muscular Dystrophy Shows That Oxidation State of Albumin Correlates with Protein Oxidation and Damage in Mdx Muscle

    A Blood Biomarker for Duchenne Muscular Dystrophy Shows That Oxidation State of Albumin Correlates with Protein Oxidation and Damage in Mdx Muscle oleh Basma A. Al-Mshhdani, Miranda D. Grounds, Peter G. Arthur, Jessica R. Terrill

    Diterbitkan 2021-08-01
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  11. 11
    Towards developing standard operating procedures for pre-clinical testing in the mdx mouse model of Duchenne muscular dystrophy

    Towards developing standard operating procedures for pre-clinical testing in the mdx mouse model of Duchenne muscular dystrophy oleh Miranda D. Grounds, Hannah G. Radley, Gordon S. Lynch, Kanneboyina Nagaraju, Annamaria De Luca

    Diterbitkan 2008-07-01
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  12. 12
    Dysferlin-deficiency has greater impact on function of slow muscles, compared with fast, in aged BLAJ mice.

    Dysferlin-deficiency has greater impact on function of slow muscles, compared with fast, in aged BLAJ mice. oleh Erin M Lloyd, Hongyang Xu, Robyn M Murphy, Miranda D Grounds, Gavin J Pinniger

    Diterbitkan 2019-01-01
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  13. 13
    Correction: Dysferlin-deficiency has greater impact on function of slow muscles, compared with fast, in aged BLAJ mice.

    Correction: Dysferlin-deficiency has greater impact on function of slow muscles, compared with fast, in aged BLAJ mice. oleh Erin M Lloyd, Hongyang Xu, Robyn M Murphy, Miranda D Grounds, Gavin J Pinniger

    Diterbitkan 2023-01-01
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  14. 14
    Beneficial effects of high dose taurine treatment in juvenile dystrophic mdx mice are offset by growth restriction.

    Beneficial effects of high dose taurine treatment in juvenile dystrophic mdx mice are offset by growth restriction. oleh Jessica R Terrill, Gavin J Pinniger, Keshav V Nair, Miranda D Grounds, Peter G Arthur

    Diterbitkan 2017-01-01
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  15. 15
    Oxidised Albumin Levels in Plasma and Skeletal Muscle as Biomarkers of Disease Progression and Treatment Efficacy in Dystrophic <i>mdx</i> Mice

    Oxidised Albumin Levels in Plasma and Skeletal Muscle as Biomarkers of Disease Progression and Treatment Efficacy in Dystrophic <i>mdx</i> Mice oleh Jessica R. Terrill, Angelo Patrick R. Bautista, Irene Tsioutsias, Miranda D. Grounds, Peter G. Arthur

    Diterbitkan 2024-06-01
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  16. 16
    Interactions between Skeletal Muscle Myoblasts and their Extracellular Matrix Revealed by a Serum Free Culture System.

    Interactions between Skeletal Muscle Myoblasts and their Extracellular Matrix Revealed by a Serum Free Culture System. oleh Vishal Chaturvedi, Danielle E Dye, Beverley F Kinnear, Toin H van Kuppevelt, Miranda D Grounds, Deirdre R Coombe

    Diterbitkan 2015-01-01
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  17. 17
    MicroRNA and Long Non-coding RNA Regulation in Skeletal Muscle From Growth to Old Age Shows Striking Dysregulation of the Callipyge Locus

    MicroRNA and Long Non-coding RNA Regulation in Skeletal Muscle From Growth to Old Age Shows Striking Dysregulation of the Callipyge Locus oleh Jasmine Mikovic, Kate Sadler, Lauren Butchart, Sarah Voisin, Frederico Gerlinger-Romero, Paul Della Gatta, Miranda D. Grounds, Séverine Lamon

    Diterbitkan 2018-11-01
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  18. 18
    Dystropathology increases energy expenditure and protein turnover in the mdx mouse model of duchenne muscular dystrophy.

    Dystropathology increases energy expenditure and protein turnover in the mdx mouse model of duchenne muscular dystrophy. oleh Hannah G Radley-Crabb, Juan C Marini, Horacio A Sosa, Liliana I Castillo, Miranda D Grounds, Marta L Fiorotto

    Diterbitkan 2014-01-01
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  19. 19
    Biomarkers for Duchenne muscular dystrophy: myonecrosis, inflammation and oxidative stress

    Biomarkers for Duchenne muscular dystrophy: myonecrosis, inflammation and oxidative stress oleh Miranda D. Grounds, Jessica R. Terrill, Basma A. Al-Mshhdani, Marisa N. Duong, Hannah G. Radley-Crabb, Peter G. Arthur

    Diterbitkan 2020-02-01
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  20. 20
    Dysferlin deficiency alters lipid metabolism and remodels the skeletal muscle lipidome in mice

    Dysferlin deficiency alters lipid metabolism and remodels the skeletal muscle lipidome in mice oleh Vanessa R. Haynes, Stacey N. Keenan, Jackie Bayliss, Erin M. Lloyd, Peter J. Meikle, Miranda D. Grounds, Matthew J. Watt

    Diterbitkan 2019-08-01
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