Hasil Pencarian - Juan Jose Poderoso

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  1. 1
    Mitochondrial Function and Nitric Oxide Utilization

    Mitochondrial Function and Nitric Oxide Utilization oleh MARIA CECILIA CARRERAS, FRANCISCO SCHÖPFER, CONSTANZA LISDERO, NATALIA RIOBÓ, JUAN JOSÉ PODEROSO

    Diterbitkan 2000-01-01
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  2. 2
    Subcellular distribution of ERK phosphorylation in tyrosine and threonine depends on redox status in murine lung cells.

    Subcellular distribution of ERK phosphorylation in tyrosine and threonine depends on redox status in murine lung cells. oleh Katia E Helfenberger, Nerina M Villalba, Bruno Buchholz, Alberto Boveris, Juan José Poderoso, Ricardo J Gelpi, Cecilia Poderoso

    Diterbitkan 2018-01-01
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  3. 3
    Akt1 intramitochondrial cycling is a crucial step in the redox modulation of cell cycle progression.

    Akt1 intramitochondrial cycling is a crucial step in the redox modulation of cell cycle progression. oleh Valeria Gabriela Antico Arciuch, Soledad Galli, María Clara Franco, Philip Y Lam, Enrique Cadenas, María Cecilia Carreras, Juan José Poderoso

    Diterbitkan 2009-10-01
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  4. 4
    New algorithm to determine true colocalization in combination with image restoration and time-lapse confocal microscopy to MAP kinases in mitochondria.

    New algorithm to determine true colocalization in combination with image restoration and time-lapse confocal microscopy to MAP kinases in mitochondria. oleh Jorge Ignacio Villalta, Soledad Galli, María Florencia Iacaruso, Valeria Gabriela Antico Arciuch, Juan José Poderoso, Elizabeth Andrea Jares-Erijman, Lía Isabel Pietrasanta

    Diterbitkan 2011-01-01
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  5. 5
    A new paradigm for MAPK: structural interactions of hERK1 with mitochondria in HeLa cells.

    A new paradigm for MAPK: structural interactions of hERK1 with mitochondria in HeLa cells. oleh Soledad Galli, Olaf Jahn, Reiner Hitt, Doerte Hesse, Lennart Opitz, Uwe Plessmann, Henning Urlaub, Juan Jose Poderoso, Elizabeth A Jares-Erijman, Thomas M Jovin

    Diterbitkan 2009-10-01
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  6. 6
    Tumor cell phenotype is sustained by selective MAPK oxidation in mitochondria.

    Tumor cell phenotype is sustained by selective MAPK oxidation in mitochondria. oleh Soledad Galli, Valeria Gabriela Antico Arciuch, Cecilia Poderoso, Daniela Paola Converso, Qiongqiong Zhou, Elisa Bal de Kier Joffé, Enrique Cadenas, Jorge Boczkowski, María Cecilia Carreras, Juan José Poderoso

    Diterbitkan 2008-06-01
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