Hasil Pencarian - Joseph A Bonanno

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  1. 1
    Mitochondrial ROS in Slc4a11 KO Corneal Endothelial Cells Lead to ER Stress

    Mitochondrial ROS in Slc4a11 KO Corneal Endothelial Cells Lead to ER Stress oleh Rajalekshmy Shyam, Diego G. Ogando, Joseph A. Bonanno

    Diterbitkan 2022-04-01
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  2. 2
    The H<sup>+</sup> Transporter SLC4A11: Roles in Metabolism, Oxidative Stress and Mitochondrial Uncoupling

    The H<sup>+</sup> Transporter SLC4A11: Roles in Metabolism, Oxidative Stress and Mitochondrial Uncoupling oleh Joseph A. Bonanno, Raji Shyam, Moonjung Choi, Diego G. Ogando

    Diterbitkan 2022-01-01
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  3. 3
    Corneal Edema in Inducible <i>Slc4a11</i> Knockout Is Initiated by Mitochondrial Superoxide Induced Src Kinase Activation

    Corneal Edema in Inducible <i>Slc4a11</i> Knockout Is Initiated by Mitochondrial Superoxide Induced Src Kinase Activation oleh Diego G. Ogando, Edward T. Kim, Shimin Li, Joseph A. Bonanno

    Diterbitkan 2023-06-01
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  4. 4
    Ammonia sensitive SLC4A11 mitochondrial uncoupling reduces glutamine induced oxidative stress

    Ammonia sensitive SLC4A11 mitochondrial uncoupling reduces glutamine induced oxidative stress oleh Diego G. Ogando, Moonjung Choi, Rajalekshmy Shyam, Shimin Li, Joseph A. Bonanno

    Diterbitkan 2019-09-01
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  5. 5
    Telomerase immortalization of human corneal endothelial cells yields functional hexagonal monolayers.

    Telomerase immortalization of human corneal endothelial cells yields functional hexagonal monolayers. oleh Thore Schmedt, Yuming Chen, Tracy T Nguyen, Shimin Li, Joseph A Bonanno, Ula V Jurkunas

    Diterbitkan 2012-01-01
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  6. 6
    Glutaminolysis is Essential for Energy Production and Ion Transport in Human Corneal Endothelium

    Glutaminolysis is Essential for Energy Production and Ion Transport in Human Corneal Endothelium oleh Wenlin Zhang, Hongde Li, Diego G. Ogando, Shimin Li, Matthew Feng, Francis W. Price, Jr, Jason M. Tennessen, Joseph A. Bonanno

    Diterbitkan 2017-02-01
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  7. 7
    Rescue of the Congenital Hereditary Endothelial Dystrophy Mouse Model by Adeno-Associated Virus–Mediated Slc4a11 Replacement

    Rescue of the Congenital Hereditary Endothelial Dystrophy Mouse Model by Adeno-Associated Virus–Mediated Slc4a11 Replacement oleh Rajalekshmy Shyam, PhD, Diego G. Ogando, PhD, Edward T. Kim, BS, Subashree Murugan, BOptom, Moonjung Choi, PhD, Joseph A. Bonanno, OD, PhD

    Diterbitkan 2022-03-01
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