Hasil Pencarian - John C. Rutledge

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  1. 1
    Lipotoxic Injury Differentially Regulates Brain Microvascular Gene Expression in Male Mice

    Lipotoxic Injury Differentially Regulates Brain Microvascular Gene Expression in Male Mice oleh Saivageethi Nuthikattu, Dragan Milenkovic, John C. Rutledge, Amparo C. Villablanca

    Diterbitkan 2020-06-01
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  2. 2
    Sex-Dependent Molecular Mechanisms of Lipotoxic Injury in Brain Microvasculature: Implications for Dementia

    Sex-Dependent Molecular Mechanisms of Lipotoxic Injury in Brain Microvasculature: Implications for Dementia oleh Saivageethi Nuthikattu, Dragan Milenkovic, John C. Rutledge, Amparo C. Villablanca

    Diterbitkan 2020-10-01
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  3. 3
    C-terminal interactions of apolipoprotein E4 respond to the postprandial state

    C-terminal interactions of apolipoprotein E4 respond to the postprandial state oleh Sarada D. Tetali, Madhu S. Budamagunta, John C. Voss, John C. Rutledge

    Diterbitkan 2006-07-01
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  4. 4
    17β-Estradiol acts separately on the LDL particle and artery wall to reduce LDL accumulation

    17β-Estradiol acts separately on the LDL particle and artery wall to reduce LDL accumulation oleh Barbara A. Walsh, Adam E. Mullick, Carole E. Banka, John C. Rutledge

    Diterbitkan 2000-01-01
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  5. 5
    17β-Estradiol reduces tumor necrosis factor-α-mediated LDL accumulation in the artery wall

    17β-Estradiol reduces tumor necrosis factor-α-mediated LDL accumulation in the artery wall oleh Barbara A. Walsh, Adam E. Mullick, Rosemary L. Walzem, John C. Rutledge

    Diterbitkan 1999-03-01
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  6. 6
    Sex-Specific Response of the Brain Free Oxylipin Profile to Soluble Epoxide Hydrolase Inhibition

    Sex-Specific Response of the Brain Free Oxylipin Profile to Soluble Epoxide Hydrolase Inhibition oleh Jennifer E. Norman, Saivageethi Nuthikattu, Dragan Milenkovic, John C. Rutledge, Amparo C. Villablanca

    Diterbitkan 2023-02-01
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  7. 7
    Triglyceride-rich lipoprotein lipolysis releases neutral and oxidized FFAs that induce endothelial cell inflammation

    Triglyceride-rich lipoprotein lipolysis releases neutral and oxidized FFAs that induce endothelial cell inflammation oleh Limin Wang, Rajan Gill, Theresa L. Pedersen, Laura J. Higgins, John W. Newman, John C. Rutledge

    Diterbitkan 2009-02-01
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  8. 8
    Angiotensin II increases vascular proteoglycan content preceding and contributing to atherosclerosis development1

    Angiotensin II increases vascular proteoglycan content preceding and contributing to atherosclerosis development1 oleh Fei Huang, Joel C. Thompson, Patricia G. Wilson, Hnin H. Aung, John C. Rutledge, Lisa R. Tannock

    Diterbitkan 2008-03-01
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  9. 9
    TGRL Lipolysis Products Induce Stress Protein ATF3 via the TGF-β Receptor Pathway in Human Aortic Endothelial Cells.

    TGRL Lipolysis Products Induce Stress Protein ATF3 via the TGF-β Receptor Pathway in Human Aortic Endothelial Cells. oleh Larissa Eiselein, Tun Nyunt, Michael W Lamé, Kit F Ng, Dennis W Wilson, John C Rutledge, Hnin H Aung

    Diterbitkan 2015-01-01
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  10. 10
    Lipotoxic brain microvascular injury is mediated by activating transcription factor 3-dependent inflammatory and oxidative stress pathways

    Lipotoxic brain microvascular injury is mediated by activating transcription factor 3-dependent inflammatory and oxidative stress pathways oleh Hnin Hnin Aung, Robin Altman, Tun Nyunt, Jeffrey Kim, Saivageethi Nuthikattu, Madhu Budamagunta, John C. Voss, Dennis Wilson, John C. Rutledge, Amparo C. Villablanca

    Diterbitkan 2016-06-01
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  11. 11
    Postprandial apoE isoform and conformational changes associated with VLDL lipolysis products modulate monocyte inflammation.

    Postprandial apoE isoform and conformational changes associated with VLDL lipolysis products modulate monocyte inflammation. oleh Laura J den Hartigh, Robin Altman, Romobia Hutchinson, Jitka Petrlova, Madhu S Budamagunta, Sarada D Tetali, Jens O Lagerstedt, John C Voss, John C Rutledge

    Diterbitkan 2012-01-01
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  12. 12
    Interaction between mild hypercholesterolemia, HDL-cholesterol levels, and angiotensin II in intimal hyperplasia in mice

    Interaction between mild hypercholesterolemia, HDL-cholesterol levels, and angiotensin II in intimal hyperplasia in mice oleh Valdeci da Cunha, Baby Martin-McNulty, Jon Vincelette, Lening Zhang, John C. Rutledge, Dennis W. Wilson, Ronald Vergona, Mark E. Sullivan, Yi-Xin Wang

    Diterbitkan 2006-03-01
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  13. 13
    VLDL lipolysis products increase VLDL fluidity and convert apolipoprotein E4 into a more expanded conformation

    VLDL lipolysis products increase VLDL fluidity and convert apolipoprotein E4 into a more expanded conformation oleh Sarada D. Tetali, Madhu S. Budamagunta, Catalina Simion, Laura J. den Hartigh, Tamás Kálai, Kálmán Hideg, Danny M. Hatters, Karl H. Weisgraber, John C. Voss, John C. Rutledge

    Diterbitkan 2010-06-01
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  14. 14
    High‐Intensity Statin Therapy Is Associated With Improved Survival in Patients With Peripheral Artery Disease

    High‐Intensity Statin Therapy Is Associated With Improved Survival in Patients With Peripheral Artery Disease oleh T. Raymond Foley, Gagan D. Singh, Damianos G. Kokkinidis, Ho‐Hin K. Choy, Tai H. Pham, Ezra A. Amsterdam, John C. Rutledge, Stephen W. Waldo, Ehrin J. Armstrong, John R. Laird

    Diterbitkan 2017-07-01
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  15. 15
    Reduced cognitive function, increased blood-brain-barrier transport and inflammatory responses, and altered brain metabolites in LDLr -/-and C57BL/6 mice fed a western diet.

    Reduced cognitive function, increased blood-brain-barrier transport and inflammatory responses, and altered brain metabolites in LDLr -/-and C57BL/6 mice fed a western diet. oleh Jennifer M Rutkowsky, Linda L Lee, Michelle Puchowicz, Mari S Golub, Douglas E Befroy, Dennis W Wilson, Steven Anderson, Gary Cline, Jason Bini, Kamil Borkowski, Trina A Knotts, John C Rutledge, Mouse Metabolic Phenotyping Center Imaging Working Group

    Diterbitkan 2018-01-01
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