Hasil Pencarian - Jan Jacob
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Modeling of Chronic Myeloid Leukemia: An Overview of In Vivo Murine and Human Xenograft Models oleh Pallavi Sontakke, Jenny Jaques, Edo Vellenga, Jan Jacob Schuringa
Diterbitkan 2016-01-01
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A Survey of Optimal Control Allocation for Aerial Vehicle Control oleh Till Martin Blaha, Ewoud Jan Jacob Smeur, Bart Diane Walter Remes
Diterbitkan 2023-07-01
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Single-cell STAT5 signal transduction profiling in normal and leukemic stem and progenitor cell populations reveals highly distinct cytokine responses. oleh Lina Han, Albertus T J Wierenga, Marjan Rozenveld-Geugien, Kim van de Lande, Edo Vellenga, Jan Jacob Schuringa
Diterbitkan 2009-01-01
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The IL1-IL1RAP axis plays an important role in the inflammatory leukemic niche that favors acute myeloid leukemia proliferation over normal hematopoiesis oleh Bauke de Boer, Sofia Sheveleva, Katja Apelt, Edo Vellenga, André B. Mulder, Gerwin Huls, Jan Jacob Schuringa
Diterbitkan 2020-10-01
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Reductions in soil surface albedo as a function of biochar application rate: implications for global radiative forcing oleh Frank G A Verheijen, Simon Jeffery, Marijn van der Velde, Vít Penížek, Martin Beland, Ana Catarina Bastos, Jan Jacob Keizer
Diterbitkan 2013-01-01
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Potential Impact of Climate Change on Suspended Sediment Yield in NW Spain: A Case Study on the Corbeira Catchment oleh M. Luz Rodríguez-Blanco, Ricardo Arias, M. Mercedes Taboada-Castro, Joao Pedro Nunes, Jan Jacob Keizer, M. Teresa Taboada-Castro
Diterbitkan 2016-10-01
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Thiostrepton induces cell death of acute myeloid leukemia blasts and the associated macrophage population oleh Isabel Weinhäuser, Diego A. Pereira-Martins, Jacobien R. Hilberink, Annet Brouwers-Vos, Eduardo M. Rego, Gerwin Huls, Jan Jacob Schuringa
Diterbitkan 2023-08-01
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BRD3/4 inhibition and FLT3-ligand deprivation target pathways that are essential for the survival of human MLL-AF9+ leukemic cells. oleh Marco Carretta, Annet Z Brouwers-Vos, Matthieu Bosman, Sarah J Horton, Joost H A Martens, Edo Vellenga, Jan Jacob Schuringa
Diterbitkan 2017-01-01
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