Hasil Pencarian - James B Bliska

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  1. 1
    The Importance of Role Models in Research.

    The Importance of Role Models in Research. oleh James B Bliska

    Diterbitkan 2016-06-01
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  2. 2
    Yersinia controls type III effector delivery into host cells by modulating Rho activity.

    Yersinia controls type III effector delivery into host cells by modulating Rho activity. oleh Edison Mejía, James B Bliska, Gloria I Viboud

    Diterbitkan 2008-01-01
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  3. 3
    The GAP activity of type III effector YopE triggers killing of Yersinia in macrophages.

    The GAP activity of type III effector YopE triggers killing of Yersinia in macrophages. oleh Xiaoying Wang, Kaustubh Parashar, Ananya Sitaram, James B Bliska

    Diterbitkan 2014-08-01
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  4. 4
    A Role for the SmpB-SsrA system in Yersinia pseudotuberculosis pathogenesis.

    A Role for the SmpB-SsrA system in Yersinia pseudotuberculosis pathogenesis. oleh Nihal A Okan, James B Bliska, A Wali Karzai

    Diterbitkan 2006-01-01
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  5. 5
    YopJ-induced caspase-1 activation in Yersinia-infected macrophages: independent of apoptosis, linked to necrosis, dispensable for innate host defense.

    YopJ-induced caspase-1 activation in Yersinia-infected macrophages: independent of apoptosis, linked to necrosis, dispensable for innate host defense. oleh Ying Zheng, Sarit Lilo, Patricio Mena, James B Bliska

    Diterbitkan 2012-01-01
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  6. 6
    <italic toggle="yes">Salmonella</italic> “Sops” Up a Preferred Electron Receptor in the Inflamed Intestine

    <italic toggle="yes">Salmonella</italic> “Sops” Up a Preferred Electron Receptor in the Inflamed Intestine oleh James B. Bliska, Adrianus W. M. van der Velden

    Diterbitkan 2012-08-01
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  7. 7
    Random mutagenesis identifies a C-terminal region of YopD important for Yersinia type III secretion function.

    Random mutagenesis identifies a C-terminal region of YopD important for Yersinia type III secretion function. oleh Rebecca Solomon, Weibing Zhang, Grace McCrann, James B Bliska, Gloria I Viboud

    Diterbitkan 2015-01-01
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  8. 8
    CCR2+ Inflammatory Dendritic Cells and Translocation of Antigen by Type III Secretion Are Required for the Exceptionally Large CD8+ T Cell Response to the Protective YopE69-77 Epitope during Yersinia Infection.

    CCR2+ Inflammatory Dendritic Cells and Translocation of Antigen by Type III Secretion Are Required for the Exceptionally Large CD8+ T Cell Response to the Protective YopE69-77 Epit... oleh Yue Zhang, Jason W Tam, Patricio Mena, Adrianus W M van der Velden, James B Bliska

    Diterbitkan 2015-10-01
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  9. 9
    A Yersinia effector with enhanced inhibitory activity on the NF-κB pathway activates the NLRP3/ASC/caspase-1 inflammasome in macrophages.

    A Yersinia effector with enhanced inhibitory activity on the NF-κB pathway activates the NLRP3/ASC/caspase-1 inflammasome in macrophages. oleh Ying Zheng, Sarit Lilo, Igor E Brodsky, Yue Zhang, Ruslan Medzhitov, Kenneth B Marcu, James B Bliska

    Diterbitkan 2011-04-01
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  10. 10
    Phosphoprotein phosphatase activity positively regulates oligomeric pyrin to trigger inflammasome assembly in phagocytes

    Phosphoprotein phosphatase activity positively regulates oligomeric pyrin to trigger inflammasome assembly in phagocytes oleh Haleema S. Malik, Flora Magnotti, Nicole A. Loeven, Jose M. Delgado, Arminja N. Kettenbach, Thomas Henry, James B. Bliska

    Diterbitkan 2023-10-01
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  11. 11
    Epidemiology of pathogenic enterobacteria in humans, livestock, and peridomestic rodents in rural Madagascar.

    Epidemiology of pathogenic enterobacteria in humans, livestock, and peridomestic rodents in rural Madagascar. oleh DeAnna C Bublitz, Patricia C Wright, Jonathan R Bodager, Fidisoa T Rasambainarivo, James B Bliska, Thomas R Gillespie

    Diterbitkan 2014-01-01
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  12. 12
    The Burkholderia cenocepacia Type VI Secretion System Effector TecA Is a Virulence Factor in Mouse Models of Lung Infection

    The Burkholderia cenocepacia Type VI Secretion System Effector TecA Is a Virulence Factor in Mouse Models of Lung Infection oleh Nicole A. Loeven, Andrew I. Perault, Peggy A. Cotter, Craig A. Hodges, Joseph D. Schwartzman, Thomas H. Hampton, James B. Bliska

    Diterbitkan 2021-10-01
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  13. 13
    IQGAP1 Is Important for Activation of Caspase-1 in Macrophages and Is Targeted by <named-content content-type="genus-species">Yersinia pestis</named-content> Type III Effector YopM

    IQGAP1 Is Important for Activation of Caspase-1 in Macrophages and Is Targeted by <named-content content-type="genus-species">Yersinia pestis</named-content> Type III Effector YopM oleh Lawton K. Chung, Naomi H. Philip, Valentina A. Schmidt, Antonius Koller, Till Strowig, Richard A. Flavell, Igor E. Brodsky, James B. Bliska

    Diterbitkan 2014-08-01
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  14. 14
    Inflammatory monocytes provide a niche for Salmonella expansion in the lumen of the inflamed intestine.

    Inflammatory monocytes provide a niche for Salmonella expansion in the lumen of the inflamed intestine. oleh Patrick A McLaughlin, Julie A Bettke, Jason W Tam, Jesse Leeds, James B Bliska, Brian P Butler, Adrianus W M van der Velden

    Diterbitkan 2019-07-01
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  15. 15
    Inflammasome Activation in Response to the Yersinia Type III Secretion System Requires Hyperinjection of Translocon Proteins YopB and YopD

    Inflammasome Activation in Response to the Yersinia Type III Secretion System Requires Hyperinjection of Translocon Proteins YopB and YopD oleh Erin E. Zwack, Annelise G. Snyder, Meghan A. Wynosky-Dolfi, Gordon Ruthel, Naomi H. Philip, Melanie M. Marketon, Matthew S. Francis, James B. Bliska, Igor E. Brodsky

    Diterbitkan 2015-02-01
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  16. 16
    TNFα and IFNγ but not perforin are critical for CD8 T cell-mediated protection against pulmonary Yersinia pestis infection.

    TNFα and IFNγ but not perforin are critical for CD8 T cell-mediated protection against pulmonary Yersinia pestis infection. oleh Frank M Szaba, Lawrence W Kummer, Debra K Duso, Ekaterina P Koroleva, Alexei V Tumanov, Andrea M Cooper, James B Bliska, Stephen T Smiley, Jr-Shiuan Lin

    Diterbitkan 2014-05-01
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  17. 17
    Biochemical, structural and molecular dynamics analyses of the potential virulence factor RipA from Yersinia pestis.

    Biochemical, structural and molecular dynamics analyses of the potential virulence factor RipA from Yersinia pestis. oleh Rodrigo Torres, Robert V Swift, Nicholas Chim, Nicole Wheatley, Benson Lan, Brian R Atwood, Céline Pujol, Banu Sankaran, James B Bliska, Rommie E Amaro, Celia W Goulding

    Diterbitkan 2011-01-01
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  18. 18
    Correction: γδ T cell IFNγ production is directly subverted by Yersinia pseudotuberculosis outer protein YopJ in mice and humans.

    Correction: γδ T cell IFNγ production is directly subverted by Yersinia pseudotuberculosis outer protein YopJ in mice and humans. oleh Timothy H Chu, Camille Khairallah, Jason Shieh, Rhea Cho, Zhijuan Qiu, Yue Zhang, Onur Eskiocak, David G Thanassi, Mark H Kaplan, Semir Beyaz, Vincent W Yang, James B Bliska, Brian S Sheridan

    Diterbitkan 2022-05-01
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  19. 19
    γδ T cell IFNγ production is directly subverted by Yersinia pseudotuberculosis outer protein YopJ in mice and humans.

    γδ T cell IFNγ production is directly subverted by Yersinia pseudotuberculosis outer protein YopJ in mice and humans. oleh Timothy H Chu, Camille Khairallah, Jason Shieh, Rhea Cho, Zhijuan Qiu, Yue Zhang, Onur Eskiocak, David G Thanassi, Mark H Kaplan, Semir Beyaz, Vincent W Yang, James B Bliska, Brian S Sheridan

    Diterbitkan 2021-12-01
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  20. 20
    γδ T cell IFNγ production is directly subverted by Yersinia pseudotuberculosis outer protein YopJ in mice and humans

    γδ T cell IFNγ production is directly subverted by Yersinia pseudotuberculosis outer protein YopJ in mice and humans oleh Timothy H. Chu, Camille Khairallah, Jason Shieh, Rhea Cho, Zhijuan Qiu, Yue Zhang, Onur Eskiocak, David G. Thanassi, Mark H. Kaplan, Semir Beyaz, Vincent W. Yang, James B. Bliska, Brian S. Sheridan

    Diterbitkan 2021-12-01
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