Hasil Pencarian - Ido Goldstein

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  1. 1
    Editorial: Regulating Liver Transcriptional Networks by Endocrine, Extracellular, and Intrinsic Cues

    Editorial: Regulating Liver Transcriptional Networks by Endocrine, Extracellular, and Intrinsic Cues oleh Lars Grøntved, Ido Goldstein

    Diterbitkan 2019-12-01
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  2. 2
    Protocol for Primary Mouse Hepatocyte Isolation

    Protocol for Primary Mouse Hepatocyte Isolation oleh Meital Charni-Natan, Ido Goldstein

    Diterbitkan 2020-09-01
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  3. 3
    Bivariate Genomic Footprinting Detects Changes in Transcription Factor Activity

    Bivariate Genomic Footprinting Detects Changes in Transcription Factor Activity oleh Songjoon Baek, Ido Goldstein, Gordon L. Hager

    Diterbitkan 2017-05-01
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  4. 4
    Synergistic gene expression during the acute phase response is characterized by transcription factor assisted loading

    Synergistic gene expression during the acute phase response is characterized by transcription factor assisted loading oleh Ido Goldstein, Ville Paakinaho, Songjoon Baek, Myong-Hee Sung, Gordon L. Hager

    Diterbitkan 2017-11-01
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  5. 5
    Fasting Hormones Synergistically Induce Amino Acid Catabolism Genes to Promote GluconeogenesisSummary

    Fasting Hormones Synergistically Induce Amino Acid Catabolism Genes to Promote GluconeogenesisSummary oleh Noga Korenfeld, Maya Finkel, Nufar Buchshtab, Meirav Bar-Shimon, Meital Charni-Natan, Ido Goldstein

    Diterbitkan 2021-01-01
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  6. 6
    Protocol for bulk and single-nuclei chromatin accessibility quantification in mouse liver tissue

    Protocol for bulk and single-nuclei chromatin accessibility quantification in mouse liver tissue oleh Noga Korenfeld, Nicolaj I. Toft, Trine V. Dam, Meital Charni-Natan, Lars Grøntved, Ido Goldstein

    Diterbitkan 2023-09-01
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  7. 7
    Mutant p53 attenuates the anti-tumorigenic activity of fibroblasts-secreted interferon beta.

    Mutant p53 attenuates the anti-tumorigenic activity of fibroblasts-secreted interferon beta. oleh Shalom Madar, Einav Harel, Ido Goldstein, Yan Stein, Ira Kogan-Sakin, Iris Kamer, Hilla Solomon, Elya Dekel, Perry Tal, Naomi Goldfinger, Gilgi Friedlander, Varda Rotter

    Diterbitkan 2013-01-01
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  8. 8
    LXR-dependent enhancer activation regulates the temporal organization of the liver's response to refeeding leading to lipogenic gene overshoot.

    LXR-dependent enhancer activation regulates the temporal organization of the liver's response to refeeding leading to lipogenic gene overshoot. oleh Noga Korenfeld, Tali Gorbonos, Maria C Romero Florian, Dan Rotaro, Dana Goldberg, Talia Radushkevitz-Frishman, Meital Charni-Natan, Meirav Bar-Shimon, Carolyn L Cummins, Ido Goldstein

    Diterbitkan 2024-09-01
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  9. 9
    The adipokine FABP4 is a key regulator of neonatal glucose homeostasis

    The adipokine FABP4 is a key regulator of neonatal glucose homeostasis oleh Idit Ron, Reut Kassif Lerner, Moran Rathaus, Rinat Livne, Sophie Ron, Ehud Barhod, Rina Hemi, Amit Tirosh, Tzipora Strauss, Keren Ofir, Ido Goldstein, Itai M. Pessach, Amir Tirosh

    Diterbitkan 2021-10-01
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  10. 10
    TMPRSS2/ERG promotes epithelial to mesenchymal transition through the ZEB1/ZEB2 axis in a prostate cancer model.

    TMPRSS2/ERG promotes epithelial to mesenchymal transition through the ZEB1/ZEB2 axis in a prostate cancer model. oleh Orit Leshem, Shalom Madar, Ira Kogan-Sakin, Iris Kamer, Ido Goldstein, Ran Brosh, Yehudit Cohen, Jasmine Jacob-Hirsch, Marcelo Ehrlich, Shmuel Ben-Sasson, Naomi Goldfinger, Ron Loewenthal, Ephraim Gazit, Varda Rotter, Raanan Berger

    Diterbitkan 2011-01-01
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  11. 11
    A novel translocation breakpoint within the BPTF gene is associated with a pre-malignant phenotype.

    A novel translocation breakpoint within the BPTF gene is associated with a pre-malignant phenotype. oleh Yosef Buganim, Ido Goldstein, Doron Lipson, Michael Milyavsky, Sylvie Polak-Charcon, Corine Mardoukh, Hilla Solomon, Eyal Kalo, Shalom Madar, Ran Brosh, Marina Perelman, Roy Navon, Naomi Goldfinger, Iris Barshack, Zohar Yakhini, Varda Rotter

    Diterbitkan 2010-01-01
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