Hasil Pencarian - David C. S. Huang

Perbaiki Hasil
  1. 1
    Rapid Selection against Truncation Mutants in Yeast Reverse Two-Hybrid Screens

    Rapid Selection against Truncation Mutants in Yeast Reverse Two-Hybrid Screens oleh Hamsa Puthalakath, Andreas Strasser, David C.S. Huang

    Diterbitkan 2001-05-01
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  2. 2
    Gefitinib-induced killing of NSCLC cell lines expressing mutant EGFR requires BIM and can be enhanced by BH3 mimetics.

    Gefitinib-induced killing of NSCLC cell lines expressing mutant EGFR requires BIM and can be enhanced by BH3 mimetics. oleh Mark S Cragg, Junya Kuroda, Hamsa Puthalakath, David C S Huang, Andreas Strasser

    Diterbitkan 2007-10-01
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  3. 3
    Co-operation of MCL-1 and BCL-XL anti-apoptotic proteins in stromal protection of MM cells from carfilzomib mediated cytotoxicity

    Co-operation of MCL-1 and BCL-XL anti-apoptotic proteins in stromal protection of MM cells from carfilzomib mediated cytotoxicity oleh Daria Galas-Filipowicz, Selina J. Chavda, Jia-Nan Gong, Jia-Nan Gong, David C. S. Huang, Asim Khwaja, Kwee Yong

    Diterbitkan 2024-04-01
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  4. 4
    MCMV-mediated inhibition of the pro-apoptotic Bak protein is required for optimal in vivo replication.

    MCMV-mediated inhibition of the pro-apoptotic Bak protein is required for optimal in vivo replication. oleh Peter Fleming, Marc Kvansakul, Valentina Voigt, Benjamin T Kile, Ruth M Kluck, David C S Huang, Mariapia A Degli-Esposti, Christopher E Andoniou

    Diterbitkan 2013-02-01
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  5. 5
    Structural basis for apoptosis inhibition by Epstein-Barr virus BHRF1.

    Structural basis for apoptosis inhibition by Epstein-Barr virus BHRF1. oleh Marc Kvansakul, Andrew H Wei, Jamie I Fletcher, Simon N Willis, Lin Chen, Andrew W Roberts, David C S Huang, Peter M Colman

    Diterbitkan 2010-12-01
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  6. 6
    A Chemical Screening Approach to Identify Novel Key Mediators of Erythroid Enucleation.

    A Chemical Screening Approach to Identify Novel Key Mediators of Erythroid Enucleation. oleh Christina B Wölwer, Luke B Pase, Helen B Pearson, Nathan J Gödde, Kurt Lackovic, David C S Huang, Sarah M Russell, Patrick O Humbert

    Diterbitkan 2015-01-01
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  7. 7
    Multiple myeloma with 1q21 amplification is highly sensitive to MCL-1 targeting

    Multiple myeloma with 1q21 amplification is highly sensitive to MCL-1 targeting oleh Anne Slomp, Laura M. Moesbergen, Jia-nan Gong, Marta Cuenca, Peter A. von dem Borne, Pieter Sonneveld, David C.S. Huang, Monique C. Minnema, Victor Peperzak

    Diterbitkan 2019-12-01
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  8. 8
    Variability of inducible expression across the hematopoietic system of tetracycline transactivator transgenic mice.

    Variability of inducible expression across the hematopoietic system of tetracycline transactivator transgenic mice. oleh Megumi Takiguchi, Lukas E Dow, Julia E Prier, Catherine L Carmichael, Benjamin T Kile, Stephen J Turner, Scott W Lowe, David C S Huang, Ross A Dickins

    Diterbitkan 2013-01-01
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  9. 9
    Identification of an activation site in Bak and mitochondrial Bax triggered by antibodies

    Identification of an activation site in Bak and mitochondrial Bax triggered by antibodies oleh Sweta Iyer, Khatira Anwari, Amber E. Alsop, Wai Shan Yuen, David C. S. Huang, John Carroll, Nicholas A. Smith, Brian J. Smith, Grant Dewson, Ruth M. Kluck

    Diterbitkan 2016-05-01
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  10. 10
    BAK/BAX-Mediated Apoptosis Is a Myc-Induced Roadblock to Reprogramming

    BAK/BAX-Mediated Apoptosis Is a Myc-Induced Roadblock to Reprogramming oleh Esther J.Y. Kim, Minna-Liisa Anko, Christoffer Flensberg, Ian J. Majewski, Fan-Suo Geng, Jaber Firas, David C.S. Huang, Mark F. van Delft, Joan K. Heath

    Diterbitkan 2018-02-01
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  11. 11
    Therapeutic Response to Non-genotoxic Activation of p53 by Nutlin3a Is Driven by PUMA-Mediated Apoptosis in Lymphoma Cells

    Therapeutic Response to Non-genotoxic Activation of p53 by Nutlin3a Is Driven by PUMA-Mediated Apoptosis in Lymphoma Cells oleh Liz J. Valente, Brandon J. Aubrey, Marco J. Herold, Gemma L. Kelly, Lina Happo, Clare L. Scott, Andrea Newbold, Ricky W. Johnstone, David C.S. Huang, Lyubomir T. Vassilev, Andreas Strasser

    Diterbitkan 2016-03-01
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  12. 12
    Replication stress induces mitotic death through parallel pathways regulated by WAPL and telomere deprotection

    Replication stress induces mitotic death through parallel pathways regulated by WAPL and telomere deprotection oleh V. Pragathi Masamsetti, Ronnie Ren Jie Low, Ka Sin Mak, Aisling O’Connor, Chris D. Riffkin, Noa Lamm, Laure Crabbe, Jan Karlseder, David C. S. Huang, Makoto T. Hayashi, Anthony J. Cesare

    Diterbitkan 2019-09-01
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  13. 13
    Key residues in the VDAC2-BAK complex can be targeted to modulate apoptosis.

    Key residues in the VDAC2-BAK complex can be targeted to modulate apoptosis. oleh Zheng Yuan, Mark F van Delft, Mark Xiang Li, Fransisca Sumardy, Brian J Smith, David C S Huang, Guillaume Lessene, Yelena Khakam, Ruitao Jin, Sitong He, Nicholas A Smith, Richard W Birkinshaw, Peter E Czabotar, Grant Dewson

    Diterbitkan 2024-05-01
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  14. 14
    Structures of BCL-2 in complex with venetoclax reveal the molecular basis of resistance mutations

    Structures of BCL-2 in complex with venetoclax reveal the molecular basis of resistance mutations oleh Richard W. Birkinshaw, Jia-nan Gong, Cindy S. Luo, Daisy Lio, Christine A. White, Mary Ann Anderson, Piers Blombery, Guillaume Lessene, Ian J. Majewski, Rachel Thijssen, Andrew W. Roberts, David C. S. Huang, Peter M. Colman, Peter E. Czabotar

    Diterbitkan 2019-06-01
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  15. 15
    Infection with flaviviruses requires BCLXL for cell survival.

    Infection with flaviviruses requires BCLXL for cell survival. oleh Tatsuya Suzuki, Toru Okamoto, Hiroshi Katoh, Yukari Sugiyama, Shinji Kusakabe, Makoto Tokunaga, Junki Hirano, Yuka Miyata, Takasuke Fukuhara, Masahito Ikawa, Takashi Satoh, Sachiyo Yoshio, Ryosuke Suzuki, Masayuki Saijo, David C S Huang, Tatsuya Kanto, Shizuo Akira, Yoshiharu Matsuura

    Diterbitkan 2018-09-01
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  16. 16
    Mesenchymal stromal cell apoptosis is required for their therapeutic function

    Mesenchymal stromal cell apoptosis is required for their therapeutic function oleh Swee Heng Milon Pang, Joshua D’Rozario, Senora Mendonca, Tejasvini Bhuvan, Natalie L. Payne, Di Zheng, Assifa Hisana, Georgia Wallis, Adele Barugahare, David Powell, Jai Rautela, Nicholas D. Huntington, Grant Dewson, David C. S. Huang, Daniel H. D. Gray, Tracy S. P. Heng

    Diterbitkan 2021-11-01
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  17. 17
    The Lck inhibitor, AMG-47a, blocks necroptosis and implicates RIPK1 in signalling downstream of MLKL

    The Lck inhibitor, AMG-47a, blocks necroptosis and implicates RIPK1 in signalling downstream of MLKL oleh Annette V. Jacobsen, Catia L. Pierotti, Kym N. Lowes, Amanda E. Au, Ying Zhang, Nima Etemadi, Cheree Fitzgibbon, Wilhelmus J. A. Kersten, André L. Samson, Mark F. van Delft, David C. S. Huang, Hélène Jousset Sabroux, Guillaume Lessene, John Silke, James M. Murphy

    Diterbitkan 2022-04-01
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  18. 18
    The SMARCA4R1157W mutation facilitates chromatin remodeling and confers PRMT1/SMARCA4 inhibitors sensitivity in colorectal cancer

    The SMARCA4R1157W mutation facilitates chromatin remodeling and confers PRMT1/SMARCA4 inhibitors sensitivity in colorectal cancer oleh Xiangwei Zeng, Bing Yao, Jianpeng Liu, Guan-Wen Gong, Ming Liu, Jiahuang Li, Hua-Feng Pan, Qixiang Li, Dongjun Yang, Peifen Lu, Dongliang Wu, Peipei Xu, Bing Chen, Panhai Chen, Ming Zhang, Ke Zen, Jian Jing, David C. S. Huang, Dijun Chen, Zhi-Wei Jiang, Quan Zhao

    Diterbitkan 2023-03-01
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  19. 19
    Correction to: PRMT1-mediated H4R3me2a recruits SMARCA4 to promote colorectal cancer progression by enhancing EGFR signaling

    Correction to: PRMT1-mediated H4R3me2a recruits SMARCA4 to promote colorectal cancer progression by enhancing EGFR signaling oleh Bing Yao, Tao Gui, Xiangwei Zeng, Yexuan Deng, Zhi Wang, Ying Wang, Dongjun Yang, Qixiang Li, Peipei Xu, Ruifeng Hu, Xinyu Li, Bing Chen, Jin Wang, Ke Zen, Haitao Li, Melissa J. Davis, Marco J. Herold, Hua-Feng Pan, Zhi-Wei Jiang, David C. S. Huang, Ming Liu, Junyi Ju, Quan Zhao

    Diterbitkan 2021-10-01
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  20. 20
    PRMT1-mediated H4R3me2a recruits SMARCA4 to promote colorectal cancer progression by enhancing EGFR signaling

    PRMT1-mediated H4R3me2a recruits SMARCA4 to promote colorectal cancer progression by enhancing EGFR signaling oleh Bing Yao, Tao Gui, Xiangwei Zeng, Yexuan Deng, Zhi Wang, Ying Wang, Dongjun Yang, Qixiang Li, Peipei Xu, Ruifeng Hu, Xinyu Li, Bing Chen, Jin Wang, Ke Zen, Haitao Li, Melissa J. Davis, Marco J. Herold, Hua-Feng Pan, Zhi-Wei Jiang, David C. S. Huang, Ming Liu, Junyi Ju, Quan Zhao

    Diterbitkan 2021-04-01
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