Hasil Pencarian - Chiranjeevi Sandi

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  1. 1
    Cellular, molecular and functional characterisation of YAC transgenic mouse models of Friedreich ataxia.

    Cellular, molecular and functional characterisation of YAC transgenic mouse models of Friedreich ataxia. oleh Sara Anjomani Virmouni, Chiranjeevi Sandi, Sahar Al-Mahdawi, Mark A Pook

    Diterbitkan 2014-01-01
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  2. 2
    Friedreich ataxia patient tissues exhibit increased 5-hydroxymethylcytosine modification and decreased CTCF binding at the FXN locus.

    Friedreich ataxia patient tissues exhibit increased 5-hydroxymethylcytosine modification and decreased CTCF binding at the FXN locus. oleh Sahar Al-Mahdawi, Chiranjeevi Sandi, Ricardo Mouro Pinto, Mark A Pook

    Diterbitkan 2013-01-01
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  3. 3
    MutLα heterodimers modify the molecular phenotype of Friedreich ataxia.

    MutLα heterodimers modify the molecular phenotype of Friedreich ataxia. oleh Vahid Ezzatizadeh, Chiranjeevi Sandi, Madhavi Sandi, Sara Anjomani-Virmouni, Sahar Al-Mahdawi, Mark A Pook

    Diterbitkan 2014-01-01
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  4. 4
    Generation and characterisation of Friedreich ataxia YG8R mouse fibroblast and neural stem cell models.

    Generation and characterisation of Friedreich ataxia YG8R mouse fibroblast and neural stem cell models. oleh Chiranjeevi Sandi, Madhavi Sandi, Harvinder Jassal, Vahid Ezzatizadeh, Sara Anjomani-Virmouni, Sahar Al-Mahdawi, Mark A Pook

    Diterbitkan 2014-01-01
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  5. 5
    A novel GAA-repeat-expansion-based mouse model of Friedreich’s ataxia

    A novel GAA-repeat-expansion-based mouse model of Friedreich’s ataxia oleh Sara Anjomani Virmouni, Vahid Ezzatizadeh, Chiranjeevi Sandi, Madhavi Sandi, Sahar Al-Mahdawi, Yogesh Chutake, Mark A. Pook

    Diterbitkan 2015-03-01
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  6. 6
    Pharmacological screening using an FXN-EGFP cellular genomic reporter assay for the therapy of Friedreich ataxia.

    Pharmacological screening using an FXN-EGFP cellular genomic reporter assay for the therapy of Friedreich ataxia. oleh Lingli Li, Lucille Voullaire, Chiranjeevi Sandi, Mark A Pook, Panos A Ioannou, Martin B Delatycki, Joseph P Sarsero

    Diterbitkan 2013-01-01
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  7. 7
    The mismatch repair system protects against intergenerational GAA repeat instability in a Friedreich ataxia mouse model

    The mismatch repair system protects against intergenerational GAA repeat instability in a Friedreich ataxia mouse model oleh Vahid Ezzatizadeh, Ricardo Mouro Pinto, Chiranjeevi Sandi, Madhavi Sandi, Sahar Al-Mahdawi, Hein te Riele, Mark A. Pook

    Diterbitkan 2012-04-01
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  8. 8
    Pms2 suppresses large expansions of the (GAA·TTC)n sequence in neuronal tissues.

    Pms2 suppresses large expansions of the (GAA·TTC)n sequence in neuronal tissues. oleh Rebecka L Bourn, Irene De Biase, Ricardo Mouro Pinto, Chiranjeevi Sandi, Sahar Al-Mahdawi, Mark A Pook, Sanjay I Bidichandani

    Diterbitkan 2012-01-01
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  9. 9
    Prolonged treatment with pimelic o-aminobenzamide HDAC inhibitors ameliorates the disease phenotype of a Friedreich ataxia mouse model

    Prolonged treatment with pimelic o-aminobenzamide HDAC inhibitors ameliorates the disease phenotype of a Friedreich ataxia mouse model oleh Chiranjeevi Sandi, Ricardo Mouro Pinto, Sahar Al-Mahdawi, Vahid Ezzatizadeh, Glenn Barnes, Steve Jones, James R. Rusche, Joel M. Gottesfeld, Mark A. Pook

    Diterbitkan 2011-06-01
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  10. 10
    Identification of potential therapeutic targets in prostate cancer through a cross‐species approach

    Identification of potential therapeutic targets in prostate cancer through a cross‐species approach oleh Sarah Jurmeister, Antonio Ramos‐Montoya, Chiranjeevi Sandi, Nelma Pértega‐Gomes, Karan Wadhwa, Alastair D Lamb, Mark J Dunning, Jan Attig, Jason S Carroll, Lee GD Fryer, Sérgio L Felisbino, David E Neal

    Diterbitkan 2018-03-01
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