Hasil Pencarian - Andy P. Tsai

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  1. 1
    Metabolic Defects Caused by High-Fat Diet Modify Disease Risk through Inflammatory and Amyloidogenic Pathways in a Mouse Model of Alzheimer’s Disease

    Metabolic Defects Caused by High-Fat Diet Modify Disease Risk through Inflammatory and Amyloidogenic Pathways in a Mouse Model of Alzheimer’s Disease oleh Austin M. Reilly, Andy P. Tsai, Peter B. Lin, Aaron C. Ericsson, Adrian L. Oblak, Hongxia Ren

    Diterbitkan 2020-09-01
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  2. 2
    Author Correction: Rare CASP6N73T variant associated with hippocampal volume exhibits decreased proteolytic activity, synaptic transmission defect, and neurodegeneration

    Author Correction: Rare CASP6N73T variant associated with hippocampal volume exhibits decreased proteolytic activity, synaptic transmission defect, and neurodegeneration oleh Libin Zhou, Kwangsik Nho, Maria G. Haddad, Nicole Cherepacha, Agne Tubeleviciute-Aydin, Andy P. Tsai, Andrew J. Saykin, P. Jesper Sjöström, Andrea C. LeBlanc

    Diterbitkan 2021-08-01
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  3. 3
    Rare CASP6N73T variant associated with hippocampal volume exhibits decreased proteolytic activity, synaptic transmission defect, and neurodegeneration

    Rare CASP6N73T variant associated with hippocampal volume exhibits decreased proteolytic activity, synaptic transmission defect, and neurodegeneration oleh Libin Zhou, Kwangsik Nho, Maria G. Haddad, Nicole Cherepacha, Agne Tubeleviciute-Aydin, Andy P. Tsai, Andrew J. Saykin, P. Jesper Sjöström, Andrea C. LeBlanc

    Diterbitkan 2021-06-01
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  4. 4
    INPP5D expression is associated with risk for Alzheimer's disease and induced by plaque-associated microglia

    INPP5D expression is associated with risk for Alzheimer's disease and induced by plaque-associated microglia oleh Andy P. Tsai, Peter Bor-Chian Lin, Chuanpeng Dong, Miguel Moutinho, Brad T. Casali, Yunlong Liu, Bruce T. Lamb, Gary E. Landreth, Adrian L. Oblak, Kwangsik Nho

    Diterbitkan 2021-06-01
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  5. 5
    Corrigendum: Age-dependent microstructure alterations in 5xFAD mice by high-resolution diffusion tensor imaging

    Corrigendum: Age-dependent microstructure alterations in 5xFAD mice by high-resolution diffusion tensor imaging oleh Surendra Maharjan, Andy P. Tsai, Peter B. Lin, Cynthia Ingraham, Megan R. Jewett, Gary E. Landreth, Gary E. Landreth, Adrian L. Oblak, Adrian L. Oblak, Nian Wang, Nian Wang

    Diterbitkan 2022-10-01
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  6. 6
    Age-dependent microstructure alterations in 5xFAD mice by high-resolution diffusion tensor imaging

    Age-dependent microstructure alterations in 5xFAD mice by high-resolution diffusion tensor imaging oleh Surendra Maharjan, Andy P. Tsai, Peter B. Lin, Cynthia Ingraham, Megan R. Jewett, Gary E. Landreth, Gary E. Landreth, Adrian L. Oblak, Adrian L. Oblak, Nian Wang, Nian Wang

    Diterbitkan 2022-08-01
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  7. 7
    PLCG2 is associated with the inflammatory response and is induced by amyloid plaques in Alzheimer’s disease

    PLCG2 is associated with the inflammatory response and is induced by amyloid plaques in Alzheimer’s disease oleh Andy P. Tsai, Chuanpeng Dong, Peter Bor-Chian Lin, Evan J. Messenger, Brad T. Casali, Miguel Moutinho, Yunlong Liu, Adrian L. Oblak, Bruce T. Lamb, Gary E. Landreth, Stephanie J. Bissel, Kwangsik Nho

    Diterbitkan 2022-02-01
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  8. 8
    TREM2 splice isoforms generate soluble TREM2 species that disrupt long-term potentiation

    TREM2 splice isoforms generate soluble TREM2 species that disrupt long-term potentiation oleh Miguel Moutinho, Israel Coronel, Andy P. Tsai, Gonzalo Viana Di Prisco, Taylor Pennington, Brady K. Atwood, Shweta S. Puntambekar, Daniel C. Smith, Pablo Martinez, Seonggyun Han, Younghee Lee, Cristian A. Lasagna-Reeves, Bruce T. Lamb, Stephanie J. Bissel, Kwangsik Nho, Gary E. Landreth

    Diterbitkan 2023-02-01
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  9. 9
    Plcg2M28L Interacts With High Fat/High Sugar Diet to Accelerate Alzheimer’s Disease-Relevant Phenotypes in Mice

    Plcg2M28L Interacts With High Fat/High Sugar Diet to Accelerate Alzheimer’s Disease-Relevant Phenotypes in Mice oleh Adrian L. Oblak, Adrian L. Oblak, Adrian L. Oblak, Kevin P. Kotredes, Ravi S. Pandey, Ravi S. Pandey, Alaina M. Reagan, Cynthia Ingraham, Cynthia Ingraham, Bridget Perkins, Bridget Perkins, Christopher Lloyd, Christopher Lloyd, Deborah Baker, Deborah Baker, Peter B. Lin, Peter B. Lin, Disha M. Soni, Disha M. Soni, Andy P. Tsai, Andy P. Tsai, Scott A. Persohn, Scott A. Persohn, Amanda A. Bedwell, Amanda A. Bedwell, Kierra Eldridge, Kierra Eldridge, Rachael Speedy, Rachael Speedy, Jill A. Meyer, Jill A. Meyer, Johnathan S. Peters, Johnathan S. Peters, Lucas L. Figueiredo, Lucas L. Figueiredo, Michael Sasner, Paul R. Territo, Paul R. Territo, Paul R. Territo, Stacey J. Sukoff Rizzo, Gregory W. Carter, Bruce T. Lamb, Bruce T. Lamb, Bruce T. Lamb, Gareth R. Howell

    Diterbitkan 2022-06-01
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  10. 10
    CYP1B1-RMDN2 Alzheimer’s disease endophenotype locus identified for cerebral tau PET

    CYP1B1-RMDN2 Alzheimer’s disease endophenotype locus identified for cerebral tau PET oleh Kwangsik Nho, Shannon L. Risacher, Liana G. Apostolova, Paula J. Bice, Jared R. Brosch, Rachael Deardorff, Kelley Faber, Martin R. Farlow, Tatiana Foroud, Sujuan Gao, Thea Rosewood, Jun Pyo Kim, Kelly Nudelman, Meichen Yu, Paul Aisen, Reisa Sperling, Basavaraj Hooli, Sergey Shcherbinin, Diana Svaldi, Clifford R. Jack, William J. Jagust, Susan Landau, Aparna Vasanthakumar, Jeffrey F. Waring, Vincent Doré, Simon M. Laws, Colin L. Masters, Tenielle Porter, Christopher C. Rowe, Victor L. Villemagne, Logan Dumitrescu, Timothy J. Hohman, Julia B. Libby, Elizabeth Mormino, Rachel F. Buckley, Keith Johnson, Hyun-Sik Yang, Ronald C. Petersen, Vijay K. Ramanan, Nilüfer Ertekin-Taner, Prashanthi Vemuri, Ann D. Cohen, Kang-Hsien Fan, M. Ilyas Kamboh, Oscar L. Lopez, David A. Bennett, Muhammad Ali, Tammie Benzinger, Carlos Cruchaga, Diana Hobbs, Philip L. De Jager, Masashi Fujita, Vaishnavi Jadhav, Bruce T. Lamb, Andy P. Tsai, Isabel Castanho, Jonathan Mill, Michael W. Weiner, for the Alzheimer’s Disease Neuroimaging Initiative (ADNI), the Department of Defense Alzheimer’s Disease Neuroimaging Initiative (DoD-ADNI), the Anti-Amyloid Treatment in Asymptomatic Alzheimer’s Study (A4 Study) and Longitudinal Evaluation of Amyloid Risk and Neurodegeneration (LEARN), the Australian Imaging, Biomarker & Lifestyle Study (AIBL), Andrew J. Saykin

    Diterbitkan 2024-09-01
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